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10.1016/j.autneu.2014.10.019 http://scihub22266oqcxt.onion/10.1016/j.autneu.2014.10.019 C4336594!4336594!25458424     free     free     free       Auton+Neurosci 2015 ; 188 (ä): 64-8 Nephropedia Template TP {{tp|p=25458424|t=2015. Functional sympatholysis in hypertension |pdf=|usr=}}{{25458424}} gab.com Text Functional sympatholysis in hypertension JO: Auton Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25458424 #FOAvip Sympathetic vasoconstriction is normally attenuated in exercising muscle by local changes in muscle metabolites and other substances that reduce vascular responsiveness to ?-adrenergic receptor activation. Termed functional sympatholysis, this protective mechanism is thought to optimize muscle blood flow distribution to match perfusion with metabolic demand. Emerging evidence from both animal and human studies indicate that functional sympatholysis is impaired in hypertension and may constitute an important underlying cause of skeletal muscle malperfusion during exercise in this common cardiovascular condition. Findings from studies of animal models of hypertension and patients with essential hypertension will be integrated in this review to provide insight into the underlying mechanisms responsible for inappropriate sympathetic vasoconstriction in exercising muscle and the treatment options that may restore functional sympatholysis and improve muscle perfusion during exercise. Twit Text FOAVip Functional sympatholysis in hypertension ????Auton Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25458424 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25458424 #FOAvip English Wikipedia <ref>{{Cite pmid|25458424}}</ref> Functional sympatholysis in hypertension #MMPMID25458424 Thomas GD Auton Neurosci 2015[Mar]; 188 (ä): 64-8 PMID25458424show ga Sympathetic vasoconstriction is normally attenuated in exercising muscle by local changes in muscle metabolites and other substances that reduce vascular responsiveness to ?-adrenergic receptor activation. Termed functional sympatholysis, this protective mechanism is thought to optimize muscle blood flow distribution to match perfusion with metabolic demand. Emerging evidence from both animal and human studies indicate that functional sympatholysis is impaired in hypertension and may constitute an important underlying cause of skeletal muscle malperfusion during exercise in this common cardiovascular condition. Findings from studies of animal models of hypertension and patients with essential hypertension will be integrated in this review to provide insight into the underlying mechanisms responsible for inappropriate sympathetic vasoconstriction in exercising muscle and the treatment options that may restore functional sympatholysis and improve muscle perfusion during exercise. äFunctional sympatholysis in hypertension (epmc).pdf DeepDyve Pubget Overpricing