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10.1161/HYPERTENSIONAHA.114.04468 http://scihub22266oqcxt.onion/10.1161/HYPERTENSIONAHA.114.04468 C4326559!4326559!25489057     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25489057&cmd=llinks): Failed to open stream: HTTP request failed! 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NULL MUTATION OF THE NADPH OXIDASE SUBUNIT p67PHOXPROTECTS THE DAHL-S RAT FROMSALT-INDUCED REDUCTIONS IN MEDULLARY BLOOD FLOW AND GFR |pdf=|usr=}}{{25489057}} gab.com Text NULL MUTATION OF THE NADPH OXIDASE SUBUNIT p67PHOXPROTECTS THE DAHL-S RAT FROMSALT-INDUCED REDUCTIONS IN MEDULLARY BLOOD FLOW AND GFR JO: Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=25489057 #FOAvip Null mutations in the p67phox subunit of NADPH-oxidase confer protection from salt-sensitivity on Dahl salt-sensitive (SS) rats. Here we track the sequential changes in medullary blood flow, glomerular filtration rate, urinary protein and mean arterial pressure in SSp67phox null rats and wild-type littermates during 21-days of 4.0% NaCl (high-salt [HS]) diet. Optical fibers were implanted in the renal medulla and medullary blood flow measured in conscious rats by laser-Doppler flowmetry. Separate groups of rats were prepared with femoral venous catheters and glomerular filtration rate measured by the transcutaneous assessment of fluorescein isothiocyanate-sinistrin disappearance curves. Mean arterial blood pressure was measured by telemetry. In wild-type rats HS caused a rapid reduction in medullary blood flow which was significantly lower than control values by HS day-6. Reduced medullary blood flow was associated with a progressive increase in mean arterial pressure, averaging 170 ± 5 mmHg by HS salt day-21. A significant reduction in glomerular filtration rate was evident at day-14 HS, after the onset of hypertension and reduced medullary blood flow. In contrast, HS had no significant effect on medullary blood flow in SSp67phox null rats and the pressor response to sodium was blunted, averaging 150 ± 3 mmHg at day-21 HS. Glomerular filtration rate was maintained throughout the study and proteinuria was reduced. In summary, when p67phox is not functional in the SS rat HS does not cause reduced medullary blood flow and salt-sensitive hypertension is attenuated, consequently renal injury is reduced and glomerular filtration rate is maintained. Twit Text FOAVip NULL MUTATION OF THE NADPH OXIDASE SUBUNIT p67PHOXPROTECTS THE DAHL-S RAT FROMSALT-INDUCED REDUCTIONS IN MEDULLARY BLOOD FLOW AND GFR ????Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=25489057 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25489057 #FOAvip English Wikipedia <ref>{{Cite pmid|25489057}}</ref> NULL MUTATION OF THE NADPH OXIDASE SUBUNIT p67PHOXPROTECTS THE DAHL-S RAT FROMSALT-INDUCED REDUCTIONS IN MEDULLARY BLOOD FLOW AND GFR #MMPMID25489057 Evans LC; Ryan RP; Broadway E; Skelton MM; Kurth T; Cowley AW Hypertension 2015[Mar]; 65 (3): 561-8 PMID25489057show ga Null mutations in the p67phox subunit of NADPH-oxidase confer protection from salt-sensitivity on Dahl salt-sensitive (SS) rats. Here we track the sequential changes in medullary blood flow, glomerular filtration rate, urinary protein and mean arterial pressure in SSp67phox null rats and wild-type littermates during 21-days of 4.0% NaCl (high-salt [HS]) diet. Optical fibers were implanted in the renal medulla and medullary blood flow measured in conscious rats by laser-Doppler flowmetry. Separate groups of rats were prepared with femoral venous catheters and glomerular filtration rate measured by the transcutaneous assessment of fluorescein isothiocyanate-sinistrin disappearance curves. Mean arterial blood pressure was measured by telemetry. In wild-type rats HS caused a rapid reduction in medullary blood flow which was significantly lower than control values by HS day-6. Reduced medullary blood flow was associated with a progressive increase in mean arterial pressure, averaging 170 ± 5 mmHg by HS salt day-21. A significant reduction in glomerular filtration rate was evident at day-14 HS, after the onset of hypertension and reduced medullary blood flow. In contrast, HS had no significant effect on medullary blood flow in SSp67phox null rats and the pressor response to sodium was blunted, averaging 150 ± 3 mmHg at day-21 HS. Glomerular filtration rate was maintained throughout the study and proteinuria was reduced. In summary, when p67phox is not functional in the SS rat HS does not cause reduced medullary blood flow and salt-sensitive hypertension is attenuated, consequently renal injury is reduced and glomerular filtration rate is maintained. äNULL MUTATION OF THE NADPH OXIDASE SUBUNIT p67PHOXPROTECTS THE DAHL-S (epmc).pdf DeepDyve Pubget Overpricing