Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.gtc.2014.11.004 http://scihub22266oqcxt.onion/10.1016/j.gtc.2014.11.004 C4324530!4324530!25667021     free     free     free       Gastroenterol+Clin+North+Am 2015 ; 44 (1): 31-8 Nephropedia Template TP {{tp|p=25667021|t=2015. Histological changes in diabetic gastroparesis |pdf=|usr=}}{{25667021}} gab.com Text Histological changes in diabetic gastroparesis JO: Gastroenterol Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=25667021 #FOAvip Our understanding of the cellular abnormalities that lead to diabetic gastroparesis has evolved in concert with our increased understanding of the key cell types that regulate gastric physiology. We now know that several key cell types are affected by diabetes, leading to gastroparesis. These changes include abnormalities in the extrinsic innervation to the stomach, loss of key neurotransmitters at the level of the enteric nervous system, smooth muscle abnormalities, loss of interstitial cells of Cajal (ICC) and most recently changes in the macrophage population resident in the muscle wall. This chapter will review our current understanding with a focus on data from human studies when available. Twit Text FOAVip Histological changes in diabetic gastroparesis ????Gastroenterol Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=25667021 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25667021 #FOAvip English Wikipedia <ref>{{Cite pmid|25667021}}</ref> Histological changes in diabetic gastroparesis #MMPMID25667021 Farrugia G Gastroenterol Clin North Am 2015[Mar]; 44 (1): 31-8 PMID25667021show ga Our understanding of the cellular abnormalities that lead to diabetic gastroparesis has evolved in concert with our increased understanding of the key cell types that regulate gastric physiology. We now know that several key cell types are affected by diabetes, leading to gastroparesis. These changes include abnormalities in the extrinsic innervation to the stomach, loss of key neurotransmitters at the level of the enteric nervous system, smooth muscle abnormalities, loss of interstitial cells of Cajal (ICC) and most recently changes in the macrophage population resident in the muscle wall. This chapter will review our current understanding with a focus on data from human studies when available. äHistological changes in diabetic gastroparesis (epmc).pdf DeepDyve Pubget Overpricing