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10.1016/j.pneurobio.2013.11.004 http://scihub22266oqcxt.onion/10.1016/j.pneurobio.2013.11.004 C4295834!4295834!24291532     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Prog+Neurobiol 2014 ; 115 (ä): 138-56 Nephropedia Template TP {{tp|p=24291532|t=2014. Vascular remodeling after ischemic stroke: mechanisms and therapeutic potentials |pdf=|usr=}}{{24291532}} gab.com Text Vascular remodeling after ischemic stroke: mechanisms and therapeutic potentials JO: Prog Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=24291532 #FOAvip The brain vasculature has been increasingly recognized as a key player that directs brain development, regulates homeostasis, and contributes to pathological processes. Following ischemic stroke, the reduction of blood flow elicits a cascade of changes and leads to vascular remodeling. However, the temporal profile of vascular changes after stroke is not well understood. Growing evidence suggests that the early phase of cerebral blood volume (CBV) increase is likely due to the improvement in collateral flow, also known as arteriogenesis, whereas the late phase of CBV increase is attributed to the surge of angiogenesis. Arteriogenesis is triggered by shear fluid stress followed by activation of endothelium and inflammatory processes, while angiogenesis induces a number of pro-angiogenic factors and circulating endothelial progenitor cells (EPCs). The status of collaterals in acute stroke has been shown to have several prognostic implications, while the causal relationship between angiogenesis and improved functional recovery has yet to be established in patients. A number of interventions aimed at enhancing cerebral blood flow including increasing collateral recruitment are under clinical investigation. Transplantation of EPCs to improve angiogenesis is also underway. Knowledge in the underlying physiological mechanisms for improved arteriogenesis and angiogenesis shall lead to more effective therapies for ischemic stroke. Twit Text FOAVip Vascular remodeling after ischemic stroke: mechanisms and therapeutic potentials ????Prog Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=24291532 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24291532 #FOAvip English Wikipedia <ref>{{Cite pmid|24291532}}</ref> Vascular remodeling after ischemic stroke: mechanisms and therapeutic potentials #MMPMID24291532 Liu J; Wang Y; Akamatsu Y; Lee CC; Stetler RA; Lawton MT; Yang GY Prog Neurobiol 2014[Apr]; 115 (ä): 138-56 PMID24291532show ga The brain vasculature has been increasingly recognized as a key player that directs brain development, regulates homeostasis, and contributes to pathological processes. Following ischemic stroke, the reduction of blood flow elicits a cascade of changes and leads to vascular remodeling. However, the temporal profile of vascular changes after stroke is not well understood. Growing evidence suggests that the early phase of cerebral blood volume (CBV) increase is likely due to the improvement in collateral flow, also known as arteriogenesis, whereas the late phase of CBV increase is attributed to the surge of angiogenesis. Arteriogenesis is triggered by shear fluid stress followed by activation of endothelium and inflammatory processes, while angiogenesis induces a number of pro-angiogenic factors and circulating endothelial progenitor cells (EPCs). The status of collaterals in acute stroke has been shown to have several prognostic implications, while the causal relationship between angiogenesis and improved functional recovery has yet to be established in patients. A number of interventions aimed at enhancing cerebral blood flow including increasing collateral recruitment are under clinical investigation. Transplantation of EPCs to improve angiogenesis is also underway. Knowledge in the underlying physiological mechanisms for improved arteriogenesis and angiogenesis shall lead to more effective therapies for ischemic stroke. äVascular remodeling after ischemic stroke: mechanisms and therapeutic (epmc).pdf DeepDyve Pubget Overpricing