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10.1165/rcmb.2014-0021TR http://scihub22266oqcxt.onion/10.1165/rcmb.2014-0021TR C4291546!4291546!24988529     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Respir+Cell+Mol+Biol 2014 ; 51 (6): 721-9 Nephropedia Template TP {{tp|p=24988529|t=2014. The Crossroads of Iron with Hypoxia and Cellular Metabolism Implications in the Pathobiology of Pulmonary Hypertension |pdf=|usr=}}{{24988529}} gab.com Text The Crossroads of Iron with Hypoxia and Cellular Metabolism Implications in the Pathobiology of Pulmonary Hypertension JO: Am J Respir Cell Mol Biol http://www.kidney.de/pget.php?&tw=1&pmid=24988529 #FOAvip The pathologic hallmark of pulmonary arterial hypertension (PAH) is pulmonary vascular remodeling, characterized by endothelial cell proliferation, smooth muscle hypertrophy, and perivascular inflammation, ultimately contributing to increased pulmonary arterial pressures. Several recent studies have observed that iron deficiency in patients with various forms of PAH is associated with worsened clinical outcome. Iron plays a key role in many cellular processes regulating the response to hypoxia, oxidative stress, cellular proliferation, and cell metabolism. Given the potential importance of iron supplementation in patients with the disease and the broad cellular functions of iron, we review its role in processes that pertain to PAH. Twit Text FOAVip The Crossroads of Iron with Hypoxia and Cellular Metabolism Implications in the Pathobiology of Pulmonary Hypertension ????Am J Respir Cell Mol Biol http://www.kidney.de/pget.php?&tw=1&pmid=24988529 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24988529 #FOAvip English Wikipedia <ref>{{Cite pmid|24988529}}</ref> The Crossroads of Iron with Hypoxia and Cellular Metabolism Implications in the Pathobiology of Pulmonary Hypertension #MMPMID24988529 Robinson JC; Graham BB; Rouault TC; Tuder RM Am J Respir Cell Mol Biol 2014[Dec]; 51 (6): 721-9 PMID24988529show ga The pathologic hallmark of pulmonary arterial hypertension (PAH) is pulmonary vascular remodeling, characterized by endothelial cell proliferation, smooth muscle hypertrophy, and perivascular inflammation, ultimately contributing to increased pulmonary arterial pressures. Several recent studies have observed that iron deficiency in patients with various forms of PAH is associated with worsened clinical outcome. Iron plays a key role in many cellular processes regulating the response to hypoxia, oxidative stress, cellular proliferation, and cell metabolism. Given the potential importance of iron supplementation in patients with the disease and the broad cellular functions of iron, we review its role in processes that pertain to PAH. äThe Crossroads of Iron with Hypoxia and Cellular Metabolism Implicati(epmc).pdf DeepDyve Pubget Overpricing