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10.1111/epi.12854 http://scihub22266oqcxt.onion/10.1111/epi.12854 C4285662!4285662 !25470332     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25470332 &cmd=llinks): Failed to open stream: HTTP request failed! 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A model of posttraumatic epilepsy after penetrating brain injuries: effect of lesion size and metal fragments |pdf=|usr=}}{{25470332 }} gab.com Text A model of posttraumatic epilepsy after penetrating brain injuries: effect of lesion size and metal fragments JO: Epilepsia http://www.kidney.de/pget.php?&tw=1&pmid=25470332 #FOAvip OBJECTIVE: Penetrating brain injury (PBI) has the highest risk for inducing posttraumatic epilepsy, and those PBIs with retained foreign materials such as bullet fragments carry the greatest risk. This study examines the potential contribution of copper, a major component of bullets, to the development of epilepsy following PBI. METHODS: Anesthetized adult male rats received a penetrating injury from the dorsal cortex to the ventral hippocampus from a high speed small bit drill. In one group of animals, copper wire was inserted into the lesion. Control animals had only the lesion or the lesion plus stainless steel wire (biologically inert foreign body). From 6 to up to 11 months following the injury the rats were monitored intermittently for the development of epilepsy with video-electroencephalography (EEG). A separate set of animals was examined for possible acute seizures in the week following the injury. RESULTS: Twenty-two of the 23 animals with copper wire developed chronic epilepsy, compared to three of the 20 control rats (lesion and lesion with stainless steel). Copper was associated with more extensive injury. The control rats with epilepsy had larger lesions. In the acute injury group, there was no difference in the incidence of seizures (83% lesion plus stainless steel, 70% lesion plus copper). SIGNIFICANCE: Copper increases the risk for epilepsy and may increase damage over time, but there were no differences between the groups in the incidence of acute postinjury seizures. Lesion size may contribute to epilepsy development in lesion-only animals. Copper may be an independent risk factor for the development of epilepsy and possible secondary injury, but lesion size also contributes to the development of epilepsy. The consequences of prolonged exposure of the brain to copper observed in these animals may have clinical implications that require further evaluation. Twit Text FOAVip A model of posttraumatic epilepsy after penetrating brain injuries: effect of lesion size and metal fragments ????Epilepsia http://www.kidney.de/pget.php?&tw=1&pmid=25470332 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25470332 #FOAvip English Wikipedia <ref>{{Cite pmid|25470332 }}</ref> A model of posttraumatic epilepsy after penetrating brain injuries: effect of lesion size and metal fragments #MMPMID25470332 Kendirli MT ; Rose DT ; Bertram EH Epilepsia 2014[Dec]; 55 (12 ): 1969-77 PMID25470332 show ga OBJECTIVE: Penetrating brain injury (PBI) has the highest risk for inducing posttraumatic epilepsy, and those PBIs with retained foreign materials such as bullet fragments carry the greatest risk. This study examines the potential contribution of copper, a major component of bullets, to the development of epilepsy following PBI. METHODS: Anesthetized adult male rats received a penetrating injury from the dorsal cortex to the ventral hippocampus from a high speed small bit drill. In one group of animals, copper wire was inserted into the lesion. Control animals had only the lesion or the lesion plus stainless steel wire (biologically inert foreign body). From 6 to up to 11 months following the injury the rats were monitored intermittently for the development of epilepsy with video-electroencephalography (EEG). A separate set of animals was examined for possible acute seizures in the week following the injury. RESULTS: Twenty-two of the 23 animals with copper wire developed chronic epilepsy, compared to three of the 20 control rats (lesion and lesion with stainless steel). Copper was associated with more extensive injury. The control rats with epilepsy had larger lesions. In the acute injury group, there was no difference in the incidence of seizures (83% lesion plus stainless steel, 70% lesion plus copper). SIGNIFICANCE: Copper increases the risk for epilepsy and may increase damage over time, but there were no differences between the groups in the incidence of acute postinjury seizures. Lesion size may contribute to epilepsy development in lesion-only animals. Copper may be an independent risk factor for the development of epilepsy and possible secondary injury, but lesion size also contributes to the development of epilepsy. The consequences of prolonged exposure of the brain to copper observed in these animals may have clinical implications that require further evaluation. |*Copper [MESH] |Animals [MESH] |Brain/*pathology [MESH] |Disease Models, Animal [MESH] |Electroencephalography [MESH] |Epilepsy/*etiology/*pathology [MESH] |Foreign Bodies/*etiology [MESH] |Head Injuries, Penetrating/*complications [MESH] |Male [MESH] |Rats [MESH] |Rats, Sprague-Dawley [MESH]A model of posttraumatic epilepsy after penetrating brain injuries: ef(epmc).pdf DeepDyve Pubget Overpricing