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10.1016/j.tins.2014.08.010 http://scihub22266oqcxt.onion/10.1016/j.tins.2014.08.010 C4253572!4253572!25236348     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Trends+Neurosci 2014 ; 37 (11): 620-8 Nephropedia Template TP {{tp|p=25236348|t=2014. Drowning stars: Reassessing the role of astrocytes in brain edema |pdf=|usr=}}{{25236348}} gab.com Text Drowning stars: Reassessing the role of astrocytes in brain edema JO: Trends Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25236348 #FOAvip Edema formation frequently complicates brain infarction, tumors and trauma. Despite the significant mortality of this condition, current treatment options are often ineffective or incompletely understood. Recent studies have revealed the existence of a brain-wide paravascular pathway for cerebrospinal (CSF) and interstitial fluid (ISF) exchange. The current review critically examines the contribution of this ?glymphatic? system to the main types of brain edema. We propose that in cytotoxic edema, energy depletion enhances glymphatic CSF influx, whilst suppressing ISF efflux. We also argue that paravascular inflammation or ?paravasculitis? plays a critical role in vasogenic edema. Finally, recent advances in diagnostic imaging of glymphatic function may hold the key to defining the edema profile of individual patients and thus enable more targeted therapy. Twit Text FOAVip Drowning stars: Reassessing the role of astrocytes in brain edema ????Trends Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25236348 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25236348 #FOAvip English Wikipedia <ref>{{Cite pmid|25236348}}</ref> Drowning stars: Reassessing the role of astrocytes in brain edema #MMPMID25236348 Thrane AS; Thrane VR; Nedergaard M Trends Neurosci 2014[Nov]; 37 (11): 620-8 PMID25236348show ga Edema formation frequently complicates brain infarction, tumors and trauma. Despite the significant mortality of this condition, current treatment options are often ineffective or incompletely understood. Recent studies have revealed the existence of a brain-wide paravascular pathway for cerebrospinal (CSF) and interstitial fluid (ISF) exchange. The current review critically examines the contribution of this ?glymphatic? system to the main types of brain edema. We propose that in cytotoxic edema, energy depletion enhances glymphatic CSF influx, whilst suppressing ISF efflux. We also argue that paravascular inflammation or ?paravasculitis? plays a critical role in vasogenic edema. Finally, recent advances in diagnostic imaging of glymphatic function may hold the key to defining the edema profile of individual patients and thus enable more targeted therapy. äDrowning stars: Reassessing the role of astrocytes in brain edema (epmc).pdf DeepDyve Pubget Overpricing