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10.1007/s11938-014-0022-y http://scihub22266oqcxt.onion/10.1007/s11938-014-0022-y C4246502!4246502!24954874     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Treat+Options+Gastroenterol 2014 ; 12 (3): 359-71 Nephropedia Template TP {{tp|p=24954874|t=2014. Genetics and Treatments Options for Recurrent Acute and Chronic Pancreatitis |pdf=|usr=}}{{24954874}} gab.com Text Genetics and Treatments Options for Recurrent Acute and Chronic Pancreatitis JO: Curr Treat Options Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=24954874 #FOAvip Worldwide research efforts demonstrate a major role of gene-environment interactions for the risk, development, and progression of most pancreatic diseases, including recurrent acute and chronic pancreatitis. New findings of pancreas disease-associated risk variants have been reported in the CPA1, GGT1, CLDN2, MMP1, MTHFR, and other genes. These risk genes and their regulatory regions must be added to the known pathogenic variants in the PRSS1, SPINK1, CFTR, CTRC, CASR, UBR1, SBDS, CEL, and CTSB genes. This new knowledge promises to improve disease management and prevention through personalized medicine. At the same time, however, knowledge of an increasing number of pathogenic variants, and their complicated effects when present in combination, results in increasing difficulty in interpretation and development of recommendations. Direct-to-consumer marketing of genetic testing results also adds complexity to disease management paradigms, especially without interpretation and, in many cases, proven accuracy. While improvements in the ability to rapidly and accurately interpret complex genetic tests are clearly needed, some results, such as pathogenic CFTR variants ? including a new class of bicarbonate-defective mutations ? and PRSS1 variants have immediate implications that direct management. In addition, discovery of pancreatitis-associated genetic variants in patients with glucose intolerance may suggest underlying type 3c diabetes, which also has implications for treatment and disease management. Twit Text FOAVip Genetics and Treatments Options for Recurrent Acute and Chronic Pancreatitis ????Curr Treat Options Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=24954874 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24954874 #FOAvip English Wikipedia <ref>{{Cite pmid|24954874}}</ref> Genetics and Treatments Options for Recurrent Acute and Chronic Pancreatitis #MMPMID24954874 Shelton CA; Whitcomb DC Curr Treat Options Gastroenterol 2014[Sep]; 12 (3): 359-71 PMID24954874show ga Worldwide research efforts demonstrate a major role of gene-environment interactions for the risk, development, and progression of most pancreatic diseases, including recurrent acute and chronic pancreatitis. New findings of pancreas disease-associated risk variants have been reported in the CPA1, GGT1, CLDN2, MMP1, MTHFR, and other genes. These risk genes and their regulatory regions must be added to the known pathogenic variants in the PRSS1, SPINK1, CFTR, CTRC, CASR, UBR1, SBDS, CEL, and CTSB genes. This new knowledge promises to improve disease management and prevention through personalized medicine. At the same time, however, knowledge of an increasing number of pathogenic variants, and their complicated effects when present in combination, results in increasing difficulty in interpretation and development of recommendations. Direct-to-consumer marketing of genetic testing results also adds complexity to disease management paradigms, especially without interpretation and, in many cases, proven accuracy. While improvements in the ability to rapidly and accurately interpret complex genetic tests are clearly needed, some results, such as pathogenic CFTR variants ? including a new class of bicarbonate-defective mutations ? and PRSS1 variants have immediate implications that direct management. In addition, discovery of pancreatitis-associated genetic variants in patients with glucose intolerance may suggest underlying type 3c diabetes, which also has implications for treatment and disease management. äGenetics and Treatments Options for Recurrent Acute and Chronic Pancre(epmc).pdf DeepDyve Pubget Overpricing