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10.1111/psyp.12346 http://scihub22266oqcxt.onion/10.1111/psyp.12346 C4231605!4231605 !25387706     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25387706 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Psychophysiology 2014 ; 51 (12 ): 1259-71 Nephropedia Template TP {{tp|p=25387706 |t=2014. Heritability and molecular genetic basis of electrodermal activity: a genome-wide association study |pdf=|usr=}}{{25387706 }} gab.com Text Heritability and molecular genetic basis of electrodermal activity: a genome-wide association study JO: Psychophysiology http://www.kidney.de/pget.php?&tw=1&pmid=25387706 #FOAvip The molecular genetic basis of electrodermal activity (EDA) was analyzed using 527,829 single nucleotide polymorphisms (SNPs) in a large population-representative sample of twins and parents (N?=?4,424) in relation to various EDA indices. Biometric analyses suggested that approximately 50% or more of variance in all EDA indices was heritable. The combined effect of all SNPs together accounted for a significant amount of variance in each index, affirming their polygenic basis and heritability. However, none of the SNPs were genome-wide significant for any EDA index. Previously reported SNP associations with disorders such as substance dependence or schizophrenia, which have been linked to EDA abnormalities, were not significant; nor were associations between EDA and genes in specific neurotransmitter systems. These results suggest that EDA is influenced by multiple genes rather than by polymorphisms with large effects. Twit Text FOAVip Heritability and molecular genetic basis of electrodermal activity: a genome-wide association study ????Psychophysiology http://www.kidney.de/pget.php?&tw=1&pmid=25387706 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25387706 #FOAvip English Wikipedia <ref>{{Cite pmid|25387706 }}</ref> Heritability and molecular genetic basis of electrodermal activity: a genome-wide association study #MMPMID25387706 Vaidyanathan U ; Isen JD ; Malone SM ; Miller MB ; McGue M ; Iacono WG Psychophysiology 2014[Dec]; 51 (12 ): 1259-71 PMID25387706 show ga The molecular genetic basis of electrodermal activity (EDA) was analyzed using 527,829 single nucleotide polymorphisms (SNPs) in a large population-representative sample of twins and parents (N?=?4,424) in relation to various EDA indices. Biometric analyses suggested that approximately 50% or more of variance in all EDA indices was heritable. The combined effect of all SNPs together accounted for a significant amount of variance in each index, affirming their polygenic basis and heritability. However, none of the SNPs were genome-wide significant for any EDA index. Previously reported SNP associations with disorders such as substance dependence or schizophrenia, which have been linked to EDA abnormalities, were not significant; nor were associations between EDA and genes in specific neurotransmitter systems. These results suggest that EDA is influenced by multiple genes rather than by polymorphisms with large effects. |*Endophenotypes [MESH] |*Polymorphism, Single Nucleotide [MESH] |Adolescent [MESH] |Female [MESH] |Galvanic Skin Response/*genetics [MESH] |Genome-Wide Association Study [MESH] |Genotype [MESH] |Humans [MESH] |Male [MESH]Heritability and molecular genetic basis of electrodermal activity: a (epmc).pdf DeepDyve Pubget Overpricing