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10.1007/s11897-014-0217-5 http://scihub22266oqcxt.onion/10.1007/s11897-014-0217-5 C4224601!4224601!25142807     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Heart+Fail+Rep 2014 ; 11 (4): 500-8 Nephropedia Template TP {{tp|p=25142807|t=2014. AUTOANTIBODIES AND CARDIOVASCULAR DYSFUNCTION: CAUSE OR CONSEQUENCE?|pdf=|usr=}}{{25142807}} gab.com Text AUTOANTIBODIES AND CARDIOVASCULAR DYSFUNCTION: CAUSE OR CONSEQUENCE JO: Curr Heart Fail Rep http://www.kidney.de/pget.php?&tw=1&pmid=25142807 #FOAvip There has been a long history of the exploration into autoimmunity as possible pathogenic factor of cardiovascular diseases from unknown cause represented by dilated cardiomyopathy (DCM). Auto-antibodies (AAbs) have emerged either as humoral responses provoked by release of "self-antigens" due to tissue damage or dysregulated humoral immunity itself. The pathogenic roles of some AAbs have been suggested by the findings from basic research using in vitro and in vivo disease model as well as clinical studies including immunoadsorption studies removing AAbs from patients with DCM. In this context, the importance of AAbs belonging to IgG3 subclass has also been implicated. In this review article we summarize the findings accumulated to date regarding AAbs which have been considered to be involved in the pathology of DCM or pregnancy-related cardiovascular disease. Furthermore, we discuss the significance of AAbs as a possible cause of DCM and the potential roles as novel therapeutic target. Twit Text FOAVip AUTOANTIBODIES AND CARDIOVASCULAR DYSFUNCTION: CAUSE OR CONSEQUENCE ????Curr Heart Fail Rep http://www.kidney.de/pget.php?&tw=1&pmid=25142807 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25142807 #FOAvip English Wikipedia <ref>{{Cite pmid|25142807}}</ref> AUTOANTIBODIES AND CARDIOVASCULAR DYSFUNCTION: CAUSE OR CONSEQUENCE? #MMPMID25142807 Nagatomo Y; Tang WHW Curr Heart Fail Rep 2014[Dec]; 11 (4): 500-8 PMID25142807show ga There has been a long history of the exploration into autoimmunity as possible pathogenic factor of cardiovascular diseases from unknown cause represented by dilated cardiomyopathy (DCM). Auto-antibodies (AAbs) have emerged either as humoral responses provoked by release of "self-antigens" due to tissue damage or dysregulated humoral immunity itself. The pathogenic roles of some AAbs have been suggested by the findings from basic research using in vitro and in vivo disease model as well as clinical studies including immunoadsorption studies removing AAbs from patients with DCM. In this context, the importance of AAbs belonging to IgG3 subclass has also been implicated. In this review article we summarize the findings accumulated to date regarding AAbs which have been considered to be involved in the pathology of DCM or pregnancy-related cardiovascular disease. Furthermore, we discuss the significance of AAbs as a possible cause of DCM and the potential roles as novel therapeutic target. äAUTOANTIBODIES AND CARDIOVASCULAR DYSFUNCTION: CAUSE OR CONSEQUENCE?(epmc).pdf DeepDyve Pubget Overpricing