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10.4161/auto.32184 http://scihub22266oqcxt.onion/10.4161/auto.32184 C4198373!4198373!25126737     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25126737&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Autophagy 2014 ; 10 (10): 1873-6 Nephropedia Template TP {{tp|p=25126737|t=2014. HMGB1-dependent and -independent autophagy |pdf=|usr=}}{{25126737}} gab.com Text HMGB1-dependent and -independent autophagy JO: Autophagy http://www.kidney.de/pget.php?&tw=1&pmid=25126737 #FOAvip HMGB1 (high mobility group box 1) is a multifunctional, ubiquitous protein located inside and outside cells that plays a critical role in various physiological and pathological processes including cell development, differentiation, inflammation, immunity, metastasis, metabolism, and death. Increasing evidence demonstrates that HMGB1-dependent autophagy promotes chemotherapy resistance, sustains tumor metabolism requirements and T cell survival, prevents polyglutamine aggregates and excitotoxicity, and protects against endotoxemia, bacterial infection, and ischemia-reperfusion injury in vitro or in vivo. In contrast, HMGB1 may not be required for autophagy in some organs such as the liver and heart. Understanding HMGB1-dependent and -independent autophagy in more detail will provide insight into the integrated stress response and guide HMGB1-based therapeutic intervention. Twit Text FOAVip HMGB1-dependent and -independent autophagy ????Autophagy http://www.kidney.de/pget.php?&tw=1&pmid=25126737 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25126737 #FOAvip English Wikipedia <ref>{{Cite pmid|25126737}}</ref> HMGB1-dependent and -independent autophagy #MMPMID25126737 Sun X; Tang D Autophagy 2014[Oct]; 10 (10): 1873-6 PMID25126737show ga HMGB1 (high mobility group box 1) is a multifunctional, ubiquitous protein located inside and outside cells that plays a critical role in various physiological and pathological processes including cell development, differentiation, inflammation, immunity, metastasis, metabolism, and death. Increasing evidence demonstrates that HMGB1-dependent autophagy promotes chemotherapy resistance, sustains tumor metabolism requirements and T cell survival, prevents polyglutamine aggregates and excitotoxicity, and protects against endotoxemia, bacterial infection, and ischemia-reperfusion injury in vitro or in vivo. In contrast, HMGB1 may not be required for autophagy in some organs such as the liver and heart. Understanding HMGB1-dependent and -independent autophagy in more detail will provide insight into the integrated stress response and guide HMGB1-based therapeutic intervention. äHMGB1-dependent and -independent autophagy (epmc).pdf DeepDyve Pubget Overpricing