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10.15252/embj.201488713 http://scihub22266oqcxt.onion/10.15252/embj.201488713 C4194117!4194117!24825350     free     free     free       EMBO+J 2014 ; 33 (12): 1287-8 Nephropedia Template TP {{tp|p=24825350|t=2014. SIRT2 controls the pentose phosphate switch |pdf=|usr=}}{{24825350}} gab.com Text SIRT2 controls the pentose phosphate switch JO: EMBO J http://www.kidney.de/pget.php?&tw=1&pmid=24825350 #FOAvip The most common enzyme defect in humans is glucose-6-phosphate dehydrogenase (G6PD) deficiency, which affects more than 400 million people. G6PD shunts glucose into the pentose phosphate pathway (PPP) to generate nucleotides and reducing potential in the form of NADPH. In this issue, Wang et al (2014) show that G6PD activity is post-translationally regulated by SIRT2, a cytoplasmic NAD+-dependent deacetylase, thereby linking NAD+ levels to DNA repair and oxidative defences, and identifying potential new approaches to treating this common genetic disease. Twit Text FOAVip SIRT2 controls the pentose phosphate switch ????EMBO J http://www.kidney.de/pget.php?&tw=1&pmid=24825350 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24825350 #FOAvip English Wikipedia <ref>{{Cite pmid|24825350}}</ref> SIRT2 controls the pentose phosphate switch #MMPMID24825350 Wu LE; Sinclair DA EMBO J 2014[Jun]; 33 (12): 1287-8 PMID24825350show ga The most common enzyme defect in humans is glucose-6-phosphate dehydrogenase (G6PD) deficiency, which affects more than 400 million people. G6PD shunts glucose into the pentose phosphate pathway (PPP) to generate nucleotides and reducing potential in the form of NADPH. In this issue, Wang et al (2014) show that G6PD activity is post-translationally regulated by SIRT2, a cytoplasmic NAD+-dependent deacetylase, thereby linking NAD+ levels to DNA repair and oxidative defences, and identifying potential new approaches to treating this common genetic disease. äSIRT2 controls the pentose phosphate switch (epmc).pdf DeepDyve Pubget Overpricing