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10.1007/s12288-014-0384-2 http://scihub22266oqcxt.onion/10.1007/s12288-014-0384-2 C4192264!4192264!25332610     free     free     free Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Indian+J+Hematol+Blood+Transfus 2014 ; 30 (Suppl 1): 325-30 Nephropedia Template TP {{tp|p=25332610|t=2014. Eosinophilic Vasculitis: Time for Recognition of a New Entity?|pdf=|usr=}}{{25332610}} gab.com Text Eosinophilic Vasculitis: Time for Recognition of a New Entity JO: Indian J Hematol Blood Transfus http://www.kidney.de/pget.php?&tw=1&pmid=25332610 #FOAvip Hypereosinophilia is part of a group of complex disorders with multisystem involvement. A 23 year old male was admitted to our centre with bilateral popliteal artery and venous thrombosis and impending gangrene of the left forefoot along with deep venous thrombosis of the right lower extremity. Investigations revealed marked peripheral blood eosinophilia (27,669/?L). Bone marrow showed increased eosinophils & eosinophil precursors and no evidence of a clonal disorder. Skin biopsy from the ulcerated lesions showed small vessel vasculitis with intense eosinophilic infiltration. Investigations to look for secondary causes of hypereosinophilia in the form of Antinuclear Antibody, P-Anti Neutrophil Cytoplasmic Antibody (ANCA) and C-ANCA and FIP1L1-PDGFRA, Bcr-Abl and JAK2V617F mutations were negative. The arterial and venous thrombosis and cutaneous vasculitis were linked to the presence of hypereosinophilic syndrome. The patient?s illness responded to high dose corticosteroids leading to complete resolution of symptoms. We reviewed the literature on the lesser known entity of eosinophilic vasculitis and its association with thrombosis. Twit Text FOAVip Eosinophilic Vasculitis: Time for Recognition of a New Entity ????Indian J Hematol Blood Transfus http://www.kidney.de/pget.php?&tw=1&pmid=25332610 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25332610 #FOAvip English Wikipedia <ref>{{Cite pmid|25332610}}</ref> Eosinophilic Vasculitis: Time for Recognition of a New Entity? #MMPMID25332610 Law AD; Varma S; Varma N; Khadwal A; Prakash G; Suri V; Kumari S; Malhotra P Indian J Hematol Blood Transfus 2014[Sep]; 30 (Suppl 1): 325-30 PMID25332610show ga Hypereosinophilia is part of a group of complex disorders with multisystem involvement. A 23 year old male was admitted to our centre with bilateral popliteal artery and venous thrombosis and impending gangrene of the left forefoot along with deep venous thrombosis of the right lower extremity. Investigations revealed marked peripheral blood eosinophilia (27,669/?L). Bone marrow showed increased eosinophils & eosinophil precursors and no evidence of a clonal disorder. Skin biopsy from the ulcerated lesions showed small vessel vasculitis with intense eosinophilic infiltration. Investigations to look for secondary causes of hypereosinophilia in the form of Antinuclear Antibody, P-Anti Neutrophil Cytoplasmic Antibody (ANCA) and C-ANCA and FIP1L1-PDGFRA, Bcr-Abl and JAK2V617F mutations were negative. The arterial and venous thrombosis and cutaneous vasculitis were linked to the presence of hypereosinophilic syndrome. The patient?s illness responded to high dose corticosteroids leading to complete resolution of symptoms. We reviewed the literature on the lesser known entity of eosinophilic vasculitis and its association with thrombosis. äEosinophilic Vasculitis: Time for Recognition of a New Entity?(epmc).pdf DeepDyve Pubget Overpricing