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10.1007/s12012-014-9252-4 http://scihub22266oqcxt.onion/10.1007/s12012-014-9252-4 C4177522!4177522!24671642     free     free     free       Cardiovasc+Toxicol 2014 ; 14 (4): 291-308 Nephropedia Template TP {{tp|p=24671642|t=2014. Alcoholic cardiomyopathy: Pathophysiologic insights |pdf=|usr=}}{{24671642}} gab.com Text Alcoholic cardiomyopathy: Pathophysiologic insights JO: Cardiovasc Toxicol http://www.kidney.de/pget.php?&tw=1&pmid=24671642 #FOAvip Alcoholic cardiomyopathy is a specific heart muscle disease found in individuals with a history of long-term heavy alcohol consumption. Alcoholic cardiomyopathy is associated with a number of adverse histological, cellular, and structural changes within the myocardium. Several mechanisms are implicated in mediating the adverse effects of ethanol, including the generation of oxidative stress, apoptotic cell death, impaired mitochondrial bioenergetics/stress, derangements in fatty acid metabolism and transport, and accelerated protein catabolism. In this review, we discuss the evidence for such mechanisms and present the potential importance of drinking patterns, genetic susceptibility, nutritional factors, race, and sex. The purpose of this review is to provide a mechanistic paradigm for future research in the area of alcoholic cardiomyopathy. Twit Text FOAVip Alcoholic cardiomyopathy: Pathophysiologic insights ????Cardiovasc Toxicol http://www.kidney.de/pget.php?&tw=1&pmid=24671642 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24671642 #FOAvip English Wikipedia <ref>{{Cite pmid|24671642}}</ref> Alcoholic cardiomyopathy: Pathophysiologic insights #MMPMID24671642 Piano MR; Phillips SA Cardiovasc Toxicol 2014[Dec]; 14 (4): 291-308 PMID24671642show ga Alcoholic cardiomyopathy is a specific heart muscle disease found in individuals with a history of long-term heavy alcohol consumption. Alcoholic cardiomyopathy is associated with a number of adverse histological, cellular, and structural changes within the myocardium. Several mechanisms are implicated in mediating the adverse effects of ethanol, including the generation of oxidative stress, apoptotic cell death, impaired mitochondrial bioenergetics/stress, derangements in fatty acid metabolism and transport, and accelerated protein catabolism. In this review, we discuss the evidence for such mechanisms and present the potential importance of drinking patterns, genetic susceptibility, nutritional factors, race, and sex. The purpose of this review is to provide a mechanistic paradigm for future research in the area of alcoholic cardiomyopathy. äAlcoholic cardiomyopathy: Pathophysiologic insights (epmc).pdf DeepDyve Pubget Overpricing