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10.1074/jbc.M114.580977 http://scihub22266oqcxt.onion/10.1074/jbc.M114.580977 C4175354!4175354!25104350     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25104350&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 280.79999999999995 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 280.79999999999995 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 280.79999999999995 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Biol+Chem 2014 ; 289 (39): 27199-214 Nephropedia Template TP {{tp|p=25104350|t=2014. Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor ? (TGF?1)-dependent Paracrine Mechanism* |pdf=|usr=}}{{25104350}} gab.com Text Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor (TGF 1)-dependent Paracrine Mechanism* JO: J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=25104350 #FOAvip Background: MicroRNA-378, a cardiomyocyte-specific miRNA, is down-regulated during heart failure.Results: miR-378 inhibition induced TGF?1 expression, which correlated with the activity of c-Fos, c-Jun, and Ras. Conditioned media of miR-378-depleted myocytes induced fibroblast activation by utilizing TGF?1-dependent paracrine mechanisms.Conclusion: miR-378 is a negative regulator of TGF?1 and cardiac fibrosis.Significance: miR-378 offers therapeutic potential for the management of heart failure. Twit Text FOAVip Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor (TGF 1)-dependent Paracrine Mechanism* ????J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=25104350 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25104350 #FOAvip English Wikipedia <ref>{{Cite pmid|25104350}}</ref> Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor ? (TGF?1)-dependent Paracrine Mechanism* #MMPMID25104350 Nagalingam RS; Sundaresan NR; Noor M; Gupta MP; Solaro RJ; Gupta M J Biol Chem 2014[Sep]; 289 (39): 27199-214 PMID25104350show ga Background: MicroRNA-378, a cardiomyocyte-specific miRNA, is down-regulated during heart failure.Results: miR-378 inhibition induced TGF?1 expression, which correlated with the activity of c-Fos, c-Jun, and Ras. Conditioned media of miR-378-depleted myocytes induced fibroblast activation by utilizing TGF?1-dependent paracrine mechanisms.Conclusion: miR-378 is a negative regulator of TGF?1 and cardiac fibrosis.Significance: miR-378 offers therapeutic potential for the management of heart failure. äDeficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the D(epmc).pdf DeepDyve Pubget Overpricing