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10.1074/jbc.M114.567628 http://scihub22266oqcxt.onion/10.1074/jbc.M114.567628 C4162191!4162191!25063808     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Biol+Chem 2014 ; 289 (37): 25925-35 Nephropedia Template TP {{tp|p=25063808|t=2014. Liver Clock Protein BMAL1 Promotes de Novo Lipogenesis through Insulin-mTORC2-AKT Signaling* |pdf=|usr=}}{{25063808}} gab.com Text Liver Clock Protein BMAL1 Promotes de Novo Lipogenesis through Insulin-mTORC2-AKT Signaling* JO: J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=25063808 #FOAvip Background: The role of BMAL1 in lipogenesis in the liver is unknown.Results: BMAL1 is necessary and required for driving lipogenic gene expression and AKT activation in vivo and in vitro in response to refeeding and insulin.Conclusion: BMAL1 promotes lipogenesis through activation of AKT and stabilization of RICTOR.Significance: BMAL1-dependent lipogenesis may be targeted for treating fatty liver disease. Twit Text FOAVip Liver Clock Protein BMAL1 Promotes de Novo Lipogenesis through Insulin-mTORC2-AKT Signaling* ????J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=25063808 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25063808 #FOAvip English Wikipedia <ref>{{Cite pmid|25063808}}</ref> Liver Clock Protein BMAL1 Promotes de Novo Lipogenesis through Insulin-mTORC2-AKT Signaling* #MMPMID25063808 Zhang D; Tong X; Arthurs B; Guha A; Rui L; Kamath A; Inoki K; Yin L J Biol Chem 2014[Sep]; 289 (37): 25925-35 PMID25063808show ga Background: The role of BMAL1 in lipogenesis in the liver is unknown.Results: BMAL1 is necessary and required for driving lipogenic gene expression and AKT activation in vivo and in vitro in response to refeeding and insulin.Conclusion: BMAL1 promotes lipogenesis through activation of AKT and stabilization of RICTOR.Significance: BMAL1-dependent lipogenesis may be targeted for treating fatty liver disease. äLiver Clock Protein BMAL1 Promotes de Novo Lipogenesis through Insulin(epmc).pdf DeepDyve Pubget Overpricing