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10.1074/jbc.M114.563312 http://scihub22266oqcxt.onion/10.1074/jbc.M114.563312 C4155675!4155675!25056949     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Biol+Chem 2014 ; 289 (36): 25088-100 Nephropedia Template TP {{tp|p=25056949|t=2014. Polyubiquitinated Tristetraprolin Protects from TNF-induced, Caspase-mediated Apoptosis* |pdf=|usr=}}{{25056949}} gab.com Text Polyubiquitinated Tristetraprolin Protects from TNF-induced, Caspase-mediated Apoptosis* JO: J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=25056949 #FOAvip Background: TNFR1 ligation activates NF-?B and JNK signals, regulating cell survival and death.Results: Tristetraprolin recruited to the TNFR1 is inducibly modified by TRAF2, resulting in prolonged JNK activation.Conclusion: TTP is involved in the balance of JNK-mediated cell survival versus death.Significance: TTP appears to be a novel regulatory component of TNFR1 signaling. Twit Text FOAVip Polyubiquitinated Tristetraprolin Protects from TNF-induced, Caspase-mediated Apoptosis* ????J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=25056949 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25056949 #FOAvip English Wikipedia <ref>{{Cite pmid|25056949}}</ref> Polyubiquitinated Tristetraprolin Protects from TNF-induced, Caspase-mediated Apoptosis* #MMPMID25056949 Resch U; Cuapio A; Sturtzel C; Hofer E; de Martin R; Holper-Schichl YM J Biol Chem 2014[Sep]; 289 (36): 25088-100 PMID25056949show ga Background: TNFR1 ligation activates NF-?B and JNK signals, regulating cell survival and death.Results: Tristetraprolin recruited to the TNFR1 is inducibly modified by TRAF2, resulting in prolonged JNK activation.Conclusion: TTP is involved in the balance of JNK-mediated cell survival versus death.Significance: TTP appears to be a novel regulatory component of TNFR1 signaling. äPolyubiquitinated Tristetraprolin Protects from TNF-induced, Caspase-m(epmc).pdf DeepDyve Pubget Overpricing