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10.1101/cshperspect.a015909 http://scihub22266oqcxt.onion/10.1101/cshperspect.a015909 C4143103!4143103!25183854     free     free     free       Cold+Spring+Harb+Perspect+Med 2014 ; 4 (9): ä Nephropedia Template TP {{tp|p=25183854|t=2014. The Genetic Basis of Aortic Aneurysm |pdf=|usr=}}{{25183854}} gab.com Text The Genetic Basis of Aortic Aneurysm JO: Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=25183854 #FOAvip Gene identification in human aortic aneurysm conditions is proceeding at a rapid pace and the integration of pathogenesis-based management strategies in clinical practice is an emerging reality. Human genetic alterations causing aneurysm involve diverse gene products including constituents of the extracellular matrix, cell surface receptors, intracellular signaling molecules, and elements of the contractile cytoskeleton. Animal modeling experiments and human genetic discoveries have extensively implicated the transforming growth factor-? (TGF-?) cytokine-signaling cascade in aneurysm progression, but mechanistic links between many gene products remain obscure. This chapter will integrate human genetic alterations associated with aortic aneurysm with current basic research findings in an attempt to form a reconciling if not unifying model for hereditary aortic aneurysm. Twit Text FOAVip The Genetic Basis of Aortic Aneurysm ????Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=25183854 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25183854 #FOAvip English Wikipedia <ref>{{Cite pmid|25183854}}</ref> The Genetic Basis of Aortic Aneurysm #MMPMID25183854 Lindsay ME; Dietz HC Cold Spring Harb Perspect Med 2014[Sep]; 4 (9): ä PMID25183854show ga Gene identification in human aortic aneurysm conditions is proceeding at a rapid pace and the integration of pathogenesis-based management strategies in clinical practice is an emerging reality. Human genetic alterations causing aneurysm involve diverse gene products including constituents of the extracellular matrix, cell surface receptors, intracellular signaling molecules, and elements of the contractile cytoskeleton. Animal modeling experiments and human genetic discoveries have extensively implicated the transforming growth factor-? (TGF-?) cytokine-signaling cascade in aneurysm progression, but mechanistic links between many gene products remain obscure. This chapter will integrate human genetic alterations associated with aortic aneurysm with current basic research findings in an attempt to form a reconciling if not unifying model for hereditary aortic aneurysm. äThe Genetic Basis of Aortic Aneurysm (epmc).pdf DeepDyve Pubget Overpricing