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10.1007/s11908-014-0407-z http://scihub22266oqcxt.onion/10.1007/s11908-014-0407-z C4112991!4112991!24819870     free     free     free       Curr+Infect+Dis+Rep 2014 ; 16 (6): 407 Nephropedia Template TP {{tp|p=24819870|t=2014. Update on Varicella Zoster Virus Vasculopathy |pdf=|usr=}}{{24819870}} gab.com Text Update on Varicella Zoster Virus Vasculopathy JO: Curr Infect Dis Rep http://www.kidney.de/pget.php?&tw=1&pmid=24819870 #FOAvip Primary infection of humans with varicella zoster virus (VZV) causes varicella (chickenpox), after which virus becomes latent in cranial nerve ganglia, dorsal root ganglia and autonomic ganglia along the entire neuraxis. As VZV-specific cell-mediated immunity declines in elderly and immunocompromised individuals, VZV reactivates from one or more ganglia and typically causes herpes zoster (shingles). Zoster may also be complicated by VZV vasculopathy due to productive virus infection of cerebral arteries. In recent decades, the clinical spectrum of VZV vasculopathy has expanded to include not only transient ischemic attacks and ischemic and hemorrhagic stroke, but also multifocal VZV vasculopathy, with temporal artery infection mimicking giant cell arteritis, extracranial vasculopathy, aneurysm with and without subarachnoid hemorrhage, arterial dissection and dolichoectasia, ischemic cranial neuropathies, cerebral venous sinus thrombosis, spinal cord infarction and peripheral thrombotic disease. Twit Text FOAVip Update on Varicella Zoster Virus Vasculopathy ????Curr Infect Dis Rep http://www.kidney.de/pget.php?&tw=1&pmid=24819870 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24819870 #FOAvip English Wikipedia <ref>{{Cite pmid|24819870}}</ref> Update on Varicella Zoster Virus Vasculopathy #MMPMID24819870 Nagel MA; Gilden D Curr Infect Dis Rep 2014[Jun]; 16 (6): 407 PMID24819870show ga Primary infection of humans with varicella zoster virus (VZV) causes varicella (chickenpox), after which virus becomes latent in cranial nerve ganglia, dorsal root ganglia and autonomic ganglia along the entire neuraxis. As VZV-specific cell-mediated immunity declines in elderly and immunocompromised individuals, VZV reactivates from one or more ganglia and typically causes herpes zoster (shingles). Zoster may also be complicated by VZV vasculopathy due to productive virus infection of cerebral arteries. In recent decades, the clinical spectrum of VZV vasculopathy has expanded to include not only transient ischemic attacks and ischemic and hemorrhagic stroke, but also multifocal VZV vasculopathy, with temporal artery infection mimicking giant cell arteritis, extracranial vasculopathy, aneurysm with and without subarachnoid hemorrhage, arterial dissection and dolichoectasia, ischemic cranial neuropathies, cerebral venous sinus thrombosis, spinal cord infarction and peripheral thrombotic disease. äUpdate on Varicella Zoster Virus Vasculopathy (epmc).pdf DeepDyve Pubget Overpricing