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10.1111/jcmm.12265 http://scihub22266oqcxt.onion/10.1111/jcmm.12265 C4112018!4112018 !24645717     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24645717 &cmd=llinks): Failed to open stream: HTTP request failed! 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Gr-1+CD11b+ myeloid cells efficiently home to site of injury after intravenous administration and enhance diabetic wound healing by neoangiogenesis |pdf=|usr=}}{{24645717 }} gab.com Text Gr-1+CD11b+ myeloid cells efficiently home to site of injury after intravenous administration and enhance diabetic wound healing by neoangiogenesis JO: J Cell Mol Med http://www.kidney.de/pget.php?&tw=1&pmid=24645717 #FOAvip Vascularization is an important factor that affects diabetic wound healing. There is increasing evidence that myeloid cell lineages play a role in neovascularization. In this study, the efficiency of Gr-1+CD11b+ myeloid cells to home to the site of injury and enhance diabetic wound healing by neoangiogenesis after intravenous administration was investigated. Gr-1+CD11b+ myeloid cells were injected into tail vein after establishment of dorsal window chamber, hindlimb ischaemia and ear-punch injury in diabetic or non-diabetic mice. The Gr-1+CD11b+ myeloid cells efficiently homed to the site of injury after intravenous administration and increased neoangiogenesis. The chemokine receptor type 4 (CXCR4) is robustly expressed by Gr-1+CD11b+ myeloid cells. Inhibition of CXCR4 decreases the homing ability of Gr-1+CD11b+ myeloid cells to the site of injury, which indicates that the CXCR4/SDF-1 axis plays an important role in the homing of Gr-1+CD11b+ myeloid cells to the site of injury. In addition, Gr-1+CD11b+ myeloid cells were found to improve blood flow recovery of ischaemic limb and enhance wound healing in diabetic mice by neoangiogenesis after intravenous administration. Taken together, the results of this study suggest that Gr-1+CD11b+ myeloid cells may serve as a potential cell therapy for diabetic wound healing. Twit Text FOAVip Gr-1+CD11b+ myeloid cells efficiently home to site of injury after intravenous administration and enhance diabetic wound healing by neoangiogenesis ????J Cell Mol Med http://www.kidney.de/pget.php?&tw=1&pmid=24645717 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24645717 #FOAvip English Wikipedia <ref>{{Cite pmid|24645717 }}</ref> Gr-1+CD11b+ myeloid cells efficiently home to site of injury after intravenous administration and enhance diabetic wound healing by neoangiogenesis #MMPMID24645717 Tong X ; Lv G ; Huang J ; Min Y ; Yang L ; Lin PC J Cell Mol Med 2014[Jun]; 18 (6 ): 1194-202 PMID24645717 show ga Vascularization is an important factor that affects diabetic wound healing. There is increasing evidence that myeloid cell lineages play a role in neovascularization. In this study, the efficiency of Gr-1+CD11b+ myeloid cells to home to the site of injury and enhance diabetic wound healing by neoangiogenesis after intravenous administration was investigated. Gr-1+CD11b+ myeloid cells were injected into tail vein after establishment of dorsal window chamber, hindlimb ischaemia and ear-punch injury in diabetic or non-diabetic mice. The Gr-1+CD11b+ myeloid cells efficiently homed to the site of injury after intravenous administration and increased neoangiogenesis. The chemokine receptor type 4 (CXCR4) is robustly expressed by Gr-1+CD11b+ myeloid cells. Inhibition of CXCR4 decreases the homing ability of Gr-1+CD11b+ myeloid cells to the site of injury, which indicates that the CXCR4/SDF-1 axis plays an important role in the homing of Gr-1+CD11b+ myeloid cells to the site of injury. In addition, Gr-1+CD11b+ myeloid cells were found to improve blood flow recovery of ischaemic limb and enhance wound healing in diabetic mice by neoangiogenesis after intravenous administration. Taken together, the results of this study suggest that Gr-1+CD11b+ myeloid cells may serve as a potential cell therapy for diabetic wound healing. |*Neovascularization, Physiologic [MESH] |*Wound Healing [MESH] |Administration, Intravenous [MESH] |Animals [MESH] |Blotting, Western [MESH] |CD11b Antigen/*metabolism [MESH] |Cells, Cultured [MESH] |Diabetes Mellitus, Experimental/metabolism/*physiopathology [MESH] |Ear/*blood supply/injuries/pathology [MESH] |Flow Cytometry [MESH] |Immunoenzyme Techniques [MESH] |Ischemia/immunology/metabolism/pathology [MESH] |Lower Extremity/*blood supply/pathology [MESH] |Mice [MESH] |Mice, Inbred C57BL [MESH] |Myeloid Cells/cytology/*immunology/metabolism [MESH] |Receptors, Chemokine/*metabolism [MESH]Gr-1+CD11b+ myeloid cells efficiently home to site of injury after int(epmc).pdf DeepDyve Pubget Overpricing