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10.1074/jbc.M113.540898 http://scihub22266oqcxt.onion/10.1074/jbc.M113.540898 C4106350!4106350!24898248     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24898248&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 278.79999999999995 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Biol+Chem 2014 ; 289 (29): 20377-85 Nephropedia Template TP {{tp|p=24898248|t=2014. NCLX Protein, but Not LETM1, Mediates Mitochondrial Ca2+ Extrusion, Thereby Limiting Ca2+-induced NAD(P)H Production and Modulating Matrix Redox State* |pdf=|usr=}}{{24898248}} gab.com Text NCLX Protein, but Not LETM1, Mediates Mitochondrial Ca2+ Extrusion, Thereby Limiting Ca2+-induced NAD(P)H Production and Modulating Matrix Redox State* JO: J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=24898248 #FOAvip Background: Whether mitochondrial Ca2+ extrusion is mediated by NCLX (mitochondrial sodium/calcium exchanger) or LETM1 (leucine zipper-EF-hand-containing transmembrane protein 1) and controls matrix redox state is unknown.Results: NCLX, but not LETM1, increases Ca2+ extrusion, limits NAD(P)H production, and promotes matrix oxidation.Conclusion: NCLX controls the duration of matrix Ca2+ elevations and their impact on redox signaling.Significance: NCLX is a potential target for the treatment of redox-dependent diseases. Twit Text FOAVip NCLX Protein, but Not LETM1, Mediates Mitochondrial Ca2+ Extrusion, Thereby Limiting Ca2+-induced NAD(P)H Production and Modulating Matrix Redox State* ????J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=24898248 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24898248 #FOAvip English Wikipedia <ref>{{Cite pmid|24898248}}</ref> NCLX Protein, but Not LETM1, Mediates Mitochondrial Ca2+ Extrusion, Thereby Limiting Ca2+-induced NAD(P)H Production and Modulating Matrix Redox State* #MMPMID24898248 De Marchi U; Santo-Domingo J; Castelbou C; Sekler I; Wiederkehr A; Demaurex N J Biol Chem 2014[Jul]; 289 (29): 20377-85 PMID24898248show ga Background: Whether mitochondrial Ca2+ extrusion is mediated by NCLX (mitochondrial sodium/calcium exchanger) or LETM1 (leucine zipper-EF-hand-containing transmembrane protein 1) and controls matrix redox state is unknown.Results: NCLX, but not LETM1, increases Ca2+ extrusion, limits NAD(P)H production, and promotes matrix oxidation.Conclusion: NCLX controls the duration of matrix Ca2+ elevations and their impact on redox signaling.Significance: NCLX is a potential target for the treatment of redox-dependent diseases. äNCLX Protein, but Not LETM1, Mediates Mitochondrial Ca2+ Extrusion, Th(epmc).pdf DeepDyve Pubget Overpricing