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10.1007/s11910-013-0374-z http://scihub22266oqcxt.onion/10.1007/s11910-013-0374-z C4051361!4051361!23884722     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Neurol+Neurosci+Rep 2013 ; 13 (9): 374 Nephropedia Template TP {{tp|p=23884722|t=2013. The Variegate Neurological Manifestations of Varicella Zoster Virus Infection |pdf=|usr=}}{{23884722}} gab.com Text The Variegate Neurological Manifestations of Varicella Zoster Virus Infection JO: Curr Neurol Neurosci Rep http://www.kidney.de/pget.php?&tw=1&pmid=23884722 #FOAvip Varicella zoster virus (VZV) is an exclusively human neurotropic alphaherpesvirus. Primary infection causes varicella (chickenpox), after which the virus becomes latent in ganglionic neurons along the entire neuraxis. With advancing age or immunosuppression, cell-mediated immunity to VZV declines, and the virus reactivates to cause zoster (shingles), dermatomal distribution, pain, and rash. Zoster is often followed by chronic pain (postherpetic neuralgia), cranial nerve palsies, zoster paresis, vasculopathy, meningoencephalitis, and multiple ocular disorders. This review covers clinical, laboratory, and pathological features of neurological complications of VZV reactivation, including diagnostic testing to verify active VZV infection in the nervous system. Additional perspectives are provided by discussions of VZV latency, animal models to study varicella pathogenesis and immunity, and of the value of vaccination of elderly individuals to boost cell-mediated immunity to VZV and prevent VZV reactivation. Twit Text FOAVip The Variegate Neurological Manifestations of Varicella Zoster Virus Infection ????Curr Neurol Neurosci Rep http://www.kidney.de/pget.php?&tw=1&pmid=23884722 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23884722 #FOAvip English Wikipedia <ref>{{Cite pmid|23884722}}</ref> The Variegate Neurological Manifestations of Varicella Zoster Virus Infection #MMPMID23884722 Gilden D; Nagel MA; Cohrs RJ; Mahalingam R Curr Neurol Neurosci Rep 2013[Sep]; 13 (9): 374 PMID23884722show ga Varicella zoster virus (VZV) is an exclusively human neurotropic alphaherpesvirus. Primary infection causes varicella (chickenpox), after which the virus becomes latent in ganglionic neurons along the entire neuraxis. With advancing age or immunosuppression, cell-mediated immunity to VZV declines, and the virus reactivates to cause zoster (shingles), dermatomal distribution, pain, and rash. Zoster is often followed by chronic pain (postherpetic neuralgia), cranial nerve palsies, zoster paresis, vasculopathy, meningoencephalitis, and multiple ocular disorders. This review covers clinical, laboratory, and pathological features of neurological complications of VZV reactivation, including diagnostic testing to verify active VZV infection in the nervous system. Additional perspectives are provided by discussions of VZV latency, animal models to study varicella pathogenesis and immunity, and of the value of vaccination of elderly individuals to boost cell-mediated immunity to VZV and prevent VZV reactivation. äThe Variegate Neurological Manifestations of Varicella Zoster Virus In(epmc).pdf DeepDyve Pubget Overpricing