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10.1016/j.semcancer.2014.01.003 http://scihub22266oqcxt.onion/10.1016/j.semcancer.2014.01.003 C4048758!4048758!24468737     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Cancer+Biol 2014 ; ä (ä): 22-9 Nephropedia Template TP {{tp|p=24468737|t=2014. Epigenetic Regulation of EBV Persistence and Oncogenesis |pdf=|usr=}}{{24468737}} gab.com Text Epigenetic Regulation of EBV Persistence and Oncogenesis JO: Semin Cancer Biol http://www.kidney.de/pget.php?&tw=1&pmid=24468737 #FOAvip Epigenetic mechanisms play a fundamental role in generating diverse and heritable patterns of viral and cellular gene expression. Epstein-Barr Virus (EBV) can adopt a variety of gene expression programs that are necessary for long-term viral persistence and latency in multiple host-cell types and conditions. The latent viral genomes assemble into chromatin structures with different histone and DNA modifications patterns that control viral gene expression. Variations in nucleosome organization and chromatin conformations can also influence gene expression by coordinating physical interactions between different regulatory elements. The viral-encoded and host-cell factors that control these epigenetic features are beginning to be understood at the genome-wide level. These epigenetic regulators can also influence viral pathogenesis by expanding tissue tropism, evading immune detection, and driving host-cell carcinogenesis. Here, we review some of the recent findings and perspectives on how the EBV epigenome plays a central role in viral latency and viral-associated carcinogenesis. Twit Text FOAVip Epigenetic Regulation of EBV Persistence and Oncogenesis ????Semin Cancer Biol http://www.kidney.de/pget.php?&tw=1&pmid=24468737 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24468737 #FOAvip English Wikipedia <ref>{{Cite pmid|24468737}}</ref> Epigenetic Regulation of EBV Persistence and Oncogenesis #MMPMID24468737 Tempera I; Lieberman PM Semin Cancer Biol 2014[Jun]; ä (ä): 22-9 PMID24468737show ga Epigenetic mechanisms play a fundamental role in generating diverse and heritable patterns of viral and cellular gene expression. Epstein-Barr Virus (EBV) can adopt a variety of gene expression programs that are necessary for long-term viral persistence and latency in multiple host-cell types and conditions. The latent viral genomes assemble into chromatin structures with different histone and DNA modifications patterns that control viral gene expression. Variations in nucleosome organization and chromatin conformations can also influence gene expression by coordinating physical interactions between different regulatory elements. The viral-encoded and host-cell factors that control these epigenetic features are beginning to be understood at the genome-wide level. These epigenetic regulators can also influence viral pathogenesis by expanding tissue tropism, evading immune detection, and driving host-cell carcinogenesis. Here, we review some of the recent findings and perspectives on how the EBV epigenome plays a central role in viral latency and viral-associated carcinogenesis. äEpigenetic Regulation of EBV Persistence and Oncogenesis (epmc).pdf DeepDyve Pubget Overpricing