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10.1038/cr.2014.65 http://scihub22266oqcxt.onion/10.1038/cr.2014.65 C4042179!4042179 !24839903     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24839903 &cmd=llinks): Failed to open stream: HTTP request failed! 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No receptor stands alone: IgG B-cell receptor intrinsic and extrinsic mechanisms contribute to antibody memory |pdf=|usr=}}{{24839903 }} gab.com Text No receptor stands alone: IgG B-cell receptor intrinsic and extrinsic mechanisms contribute to antibody memory JO: Cell Res http://www.kidney.de/pget.php?&tw=1&pmid=24839903 #FOAvip Acquired immunological memory is a striking phenomenon. A lethal epidemic sweeps through a naïve population, many die but those who survive are never "attacked twice - never at least fatally", as the historian Thucydides observed in 430 BCE. Antibody memory is critical for protection against many human infectious diseases and is the basis for nearly all current human vaccines. Antibody memory is encoded, in part, in isotype-switched immunoglobulin (Ig)G-expressing memory B cells that are generated in the primary response to antigen and give rise to rapid, high-affinity and high-titered antibody responses upon challenge with the same antigen. How IgG-B-cell receptors (BCRs) and antigen-induced IgG-BCR signaling contribute to memory antibody responses are not fully understood. In this review, we summarize exciting new advances that are revealing the cellular and molecular mechanisms at play in antibody memory and discuss how studies using different experimental approaches will help elucidate the complex phenomenon of B-cell memory. Twit Text FOAVip No receptor stands alone: IgG B-cell receptor intrinsic and extrinsic mechanisms contribute to antibody memory ????Cell Res http://www.kidney.de/pget.php?&tw=1&pmid=24839903 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24839903 #FOAvip English Wikipedia <ref>{{Cite pmid|24839903 }}</ref> No receptor stands alone: IgG B-cell receptor intrinsic and extrinsic mechanisms contribute to antibody memory #MMPMID24839903 Xu Y ; Xu L ; Zhao M ; Xu C ; Fan Y ; Pierce SK ; Liu W Cell Res 2014[Jun]; 24 (6 ): 651-64 PMID24839903 show ga Acquired immunological memory is a striking phenomenon. A lethal epidemic sweeps through a naïve population, many die but those who survive are never "attacked twice - never at least fatally", as the historian Thucydides observed in 430 BCE. Antibody memory is critical for protection against many human infectious diseases and is the basis for nearly all current human vaccines. Antibody memory is encoded, in part, in isotype-switched immunoglobulin (Ig)G-expressing memory B cells that are generated in the primary response to antigen and give rise to rapid, high-affinity and high-titered antibody responses upon challenge with the same antigen. How IgG-B-cell receptors (BCRs) and antigen-induced IgG-BCR signaling contribute to memory antibody responses are not fully understood. In this review, we summarize exciting new advances that are revealing the cellular and molecular mechanisms at play in antibody memory and discuss how studies using different experimental approaches will help elucidate the complex phenomenon of B-cell memory. |Animals [MESH] |Antibodies/*immunology/metabolism [MESH] |B-Lymphocytes/cytology/immunology/metabolism [MESH] |GRB2 Adaptor Protein/genetics/metabolism [MESH] |Humans [MESH] |Immunoglobulin G/metabolism [MESH] |Receptors, Antigen, B-Cell/chemistry/*metabolism [MESH]No receptor stands alone: IgG B-cell receptor intrinsic and extrinsic (epmc).pdf DeepDyve Pubget Overpricing