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10.1089/ars.2013.5608 http://scihub22266oqcxt.onion/10.1089/ars.2013.5608 C4026303!4026303!24093231     free     free     free       Antioxid+Redox+Signal 2014 ; 20 (17): 2838-53 Nephropedia Template TP {{tp|p=24093231|t=2014. NADPH Oxidases in Lung Health and Disease |pdf=|usr=}}{{24093231}} gab.com Text NADPH Oxidases in Lung Health and Disease JO: Antioxid Redox Signal http://www.kidney.de/pget.php?&tw=1&pmid=24093231 #FOAvip Significance: The evolution of the lungs and circulatory systems in vertebrates ensured the availability of molecular oxygen (O2; dioxygen) for aerobic cellular metabolism of internal organs in large animals. O2 serves as the physiologic terminal acceptor of mitochondrial electron transfer and of the NADPH oxidase (Nox) family of oxidoreductases to generate primarily water and reactive oxygen species (ROS), respectively. Recent advances: The purposeful generation of ROS by Nox family enzymes suggests important roles in normal physiology and adaptation, most notably in host defense against invading pathogens and in cellular signaling. Critical issues: However, there is emerging evidence that, in the context of chronic stress and/or aging, Nox enzymes contribute to the pathogenesis of a number of lung diseases. Future Directions: Here, we review evolving functions of Nox enzymes in normal lung physiology and emerging pathophysiologic roles in lung disease. Antioxid. Redox Signal. 20, 2838?2853. Twit Text FOAVip NADPH Oxidases in Lung Health and Disease ????Antioxid Redox Signal http://www.kidney.de/pget.php?&tw=1&pmid=24093231 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24093231 #FOAvip English Wikipedia <ref>{{Cite pmid|24093231}}</ref> NADPH Oxidases in Lung Health and Disease #MMPMID24093231 Bernard K; Hecker L; Luckhardt TR; Cheng G; Thannickal VJ Antioxid Redox Signal 2014[Jun]; 20 (17): 2838-53 PMID24093231show ga Significance: The evolution of the lungs and circulatory systems in vertebrates ensured the availability of molecular oxygen (O2; dioxygen) for aerobic cellular metabolism of internal organs in large animals. O2 serves as the physiologic terminal acceptor of mitochondrial electron transfer and of the NADPH oxidase (Nox) family of oxidoreductases to generate primarily water and reactive oxygen species (ROS), respectively. Recent advances: The purposeful generation of ROS by Nox family enzymes suggests important roles in normal physiology and adaptation, most notably in host defense against invading pathogens and in cellular signaling. Critical issues: However, there is emerging evidence that, in the context of chronic stress and/or aging, Nox enzymes contribute to the pathogenesis of a number of lung diseases. Future Directions: Here, we review evolving functions of Nox enzymes in normal lung physiology and emerging pathophysiologic roles in lung disease. Antioxid. Redox Signal. 20, 2838?2853. äNADPH Oxidases in Lung Health and Disease (epmc).pdf DeepDyve Pubget Overpricing