Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1101/cshperspect.a018358 http://scihub22266oqcxt.onion/10.1101/cshperspect.a018358 C3996471!3996471!24789822     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cold+Spring+Harb+Perspect+Biol 2014 ; 6 (5): ä Nephropedia Template TP {{tp|p=24789822|t=2014. Endocytosis and Autophagy: Exploitation or Cooperation?|pdf=|usr=}}{{24789822}} gab.com Text Endocytosis and Autophagy: Exploitation or Cooperation JO: Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=24789822 #FOAvip Autophagy is a lysosome-mediated degradative system that is a highly conserved pathway present in all eukaryotes. In all cells, double-membrane autophagosomes form and engulf cytoplasmic components, delivering them to the lysosome for degradation. Autophagy is essential for cell health and can be activated to function as a recycling pathway in the absence of nutrients or as a quality-control pathway to eliminate damaged organelles or even to eliminate invading pathogens. Autophagy was first identified as a pathway in mammalian cells using morphological techniques, but the Atg (autophagy-related) genes required for autophagy were identified in yeast genetic screens. Despite tremendous advances in elucidating the function of individual Atg proteins, our knowledge of how autophagosomes form and subsequently interact with the endosomal pathway has lagged behind. Recent progress toward understanding where and how both the endocytotic and autophagic pathways overlap is reviewed here. Twit Text FOAVip Endocytosis and Autophagy: Exploitation or Cooperation ????Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=24789822 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24789822 #FOAvip English Wikipedia <ref>{{Cite pmid|24789822}}</ref> Endocytosis and Autophagy: Exploitation or Cooperation? #MMPMID24789822 Tooze SA; Abada A; Elazar Z Cold Spring Harb Perspect Biol 2014[May]; 6 (5): ä PMID24789822show ga Autophagy is a lysosome-mediated degradative system that is a highly conserved pathway present in all eukaryotes. In all cells, double-membrane autophagosomes form and engulf cytoplasmic components, delivering them to the lysosome for degradation. Autophagy is essential for cell health and can be activated to function as a recycling pathway in the absence of nutrients or as a quality-control pathway to eliminate damaged organelles or even to eliminate invading pathogens. Autophagy was first identified as a pathway in mammalian cells using morphological techniques, but the Atg (autophagy-related) genes required for autophagy were identified in yeast genetic screens. Despite tremendous advances in elucidating the function of individual Atg proteins, our knowledge of how autophagosomes form and subsequently interact with the endosomal pathway has lagged behind. Recent progress toward understanding where and how both the endocytotic and autophagic pathways overlap is reviewed here. äEndocytosis and Autophagy: Exploitation or Cooperation?(epmc).pdf DeepDyve Pubget Overpricing