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.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117 J+Adolesc+Health
2014 ; 54
(4
): 435-441.e2
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Understanding the link between early sexual initiation and later sexually
transmitted infection: test and replication in two longitudinal studies
#MMPMID24280303
Epstein M
; Bailey JA
; Manhart LE
; Hill KG
; Hawkins JD
; Haggerty KP
; Catalano RF
J Adolesc Health
2014[Apr]; 54
(4
): 435-441.e2
PMID24280303
show ga
PURPOSE: Age at sexual initiation is strongly associated with sexually
transmitted infections (STI); yet, prevention programs aiming to delay sexual
initiation have shown mixed results in reducing STI. This study tested three
explanatory mechanisms for the relationship between early sexual debut and STI:
number of sexual partners, individual characteristics, and environmental
antecedents. METHODS: A test-and-replicate strategy was employed using two
longitudinal studies: the Seattle Social Development Project (SSDP) and Raising
Healthy Children (RHC). Childhood measures included pubertal age, behavioral
disinhibition, and family, school, and peer influences. Alcohol use and age of
sexual debut were measured during adolescence. Lifetime number of sexual partners
and having sex under the influence were measured during young adulthood. Sexually
transmitted infection diagnosis was self-reported at age 24. Early sex was
defined as debut at <15 years. Path models were developed in SSDP evaluating
relationships between measures, and were then tested in RHC. RESULTS: The
relationship between early sex and STI was fully mediated by lifetime sex
partners in SSDP, but only partially in RHC, after accounting for co-occurring
factors. Behavioral disinhibition predicted early sex, early alcohol use, number
of sexual partners, and sex under the influence, but had no direct effect on STI.
Family management protected against early sex and early alcohol use, whereas
antisocial peers exacerbated the risk. CONCLUSIONS: Early sexual initiation, a
key mediator of STI, is driven by antecedents that influence multiple risk
behaviors. Targeting co-occurring individual and environmental factors may be
more effective than discouraging early sexual debut and may concomitantly improve
other risk behaviors.