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10.1007/s10753-013-9668-1 http://scihub22266oqcxt.onion/10.1007/s10753-013-9668-1 C3825492!3825492!23807051     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Inflammation 2013 ; 36 (6): 1295-303 Nephropedia Template TP {{tp|p=23807051|t=2013. MHV68 Latency Modulates the Host Immune Response to Influenza A Virus |pdf=|usr=}}{{23807051}} gab.com Text MHV68 Latency Modulates the Host Immune Response to Influenza A Virus JO: Inflammation http://www.kidney.de/pget.php?&tw=1&pmid=23807051 #FOAvip Murine gammaherpesvirus 68 (MHV68) is a natural rodent pathogen that has been used as a model to study the pathogenesis of human gammaherpesviruses. Like other herpesviruses, MHV68 causes acute infection and establishes life-long latency in the host. Recently, it has been shown that mice latently infected with MHV68 have resistance to unrelated pathogens in secondary infection models. We therefore hypothesized that latent MHV68 infection could modulate the host response to influenza A virus. To test this hypothesis, mice were infected intranasally with influenza virus following the establishment of MHV68 latency. Mice latently infected with MHV68 showed significantly higher survival to influenza A virus infection than did PBS mock-infected mice. Latent MHV68 infection led to lower influenza viral loads and decreased inflammatory pathology in the lungs. Alveolar macrophages of mice latently infected with MHV68 showed activated status, and adoptive transfer of those activated macrophages into mice followed the infection with influenza A virus had significantly greater survival rates than control mice, suggesting that activated alveolar macrophages are a key mechanistic component in protection from secondary infections. Twit Text FOAVip MHV68 Latency Modulates the Host Immune Response to Influenza A Virus ????Inflammation http://www.kidney.de/pget.php?&tw=1&pmid=23807051 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23807051 #FOAvip English Wikipedia <ref>{{Cite pmid|23807051}}</ref> MHV68 Latency Modulates the Host Immune Response to Influenza A Virus #MMPMID23807051 Saito F; Ito T; Connett JM; Schaller MA; Carson WF; Hogaboam CM; Rochford R; Kunkel SL Inflammation 2013[]; 36 (6): 1295-303 PMID23807051show ga Murine gammaherpesvirus 68 (MHV68) is a natural rodent pathogen that has been used as a model to study the pathogenesis of human gammaherpesviruses. Like other herpesviruses, MHV68 causes acute infection and establishes life-long latency in the host. Recently, it has been shown that mice latently infected with MHV68 have resistance to unrelated pathogens in secondary infection models. We therefore hypothesized that latent MHV68 infection could modulate the host response to influenza A virus. To test this hypothesis, mice were infected intranasally with influenza virus following the establishment of MHV68 latency. Mice latently infected with MHV68 showed significantly higher survival to influenza A virus infection than did PBS mock-infected mice. Latent MHV68 infection led to lower influenza viral loads and decreased inflammatory pathology in the lungs. Alveolar macrophages of mice latently infected with MHV68 showed activated status, and adoptive transfer of those activated macrophages into mice followed the infection with influenza A virus had significantly greater survival rates than control mice, suggesting that activated alveolar macrophages are a key mechanistic component in protection from secondary infections. äMHV68 Latency Modulates the Host Immune Response to Influenza A Virus (epmc).pdf DeepDyve Pubget Overpricing