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10.1002/jbmr.1993 http://scihub22266oqcxt.onion/10.1002/jbmr.1993 C3792843!3792843!23703881     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=23703881&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Bone+Miner+Res 2013 ; 28 (12): ä Nephropedia Template TP {{tp|p=23703881|t=2013. Kidney Stones: A Fetal Origins Hypothesis? |pdf=|usr=}}{{23703881}} gab.com Text Kidney Stones: A Fetal Origins Hypothesis JO: J Bone Miner Res http://www.kidney.de/pget.php?&tw=1&pmid=23703881 #FOAvip Kidney stones are common with a multifactorial aetiology involving dietary, environmental and genetic factors. In addition, patients with nephrolithiasis are at greater risk of hypertension, diabetes, metabolic syndrome, and osteoporosis although the basis for this is not fully understood. All of these renal stone associated conditions have also been linked with adverse early life events, including low birth weight, and it has been suggested that this developmental effect is due to excess exposure to maternal glucocorticoids in utero. This is proposed to result in long-term increased hypothalamic-pituitary-axis activation and there are mechanisms through which this effect could also promote urinary lithogenic potential. We therefore hypothesise that the association between renal stone disease and hypertension, diabetes, metabolic syndrome and osteoporosis may be related by a common pathway of programming in early life which, if validated, would implicate the developmental origins hypothesis in the aetiology of nephrolithiasis. Twit Text FOAVip Kidney Stones: A Fetal Origins Hypothesis ????J Bone Miner Res http://www.kidney.de/pget.php?&tw=1&pmid=23703881 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23703881 #FOAvip English Wikipedia <ref>{{Cite pmid|23703881}}</ref> Kidney Stones: A Fetal Origins Hypothesis? #MMPMID23703881 Howles SA; Edwards MH; Cooper C; Thakker RV J Bone Miner Res 2013[Dec]; 28 (12): ä PMID23703881show ga Kidney stones are common with a multifactorial aetiology involving dietary, environmental and genetic factors. In addition, patients with nephrolithiasis are at greater risk of hypertension, diabetes, metabolic syndrome, and osteoporosis although the basis for this is not fully understood. All of these renal stone associated conditions have also been linked with adverse early life events, including low birth weight, and it has been suggested that this developmental effect is due to excess exposure to maternal glucocorticoids in utero. This is proposed to result in long-term increased hypothalamic-pituitary-axis activation and there are mechanisms through which this effect could also promote urinary lithogenic potential. We therefore hypothesise that the association between renal stone disease and hypertension, diabetes, metabolic syndrome and osteoporosis may be related by a common pathway of programming in early life which, if validated, would implicate the developmental origins hypothesis in the aetiology of nephrolithiasis. äKidney Stones: A Fetal Origins Hypothesis? (epmc).pdf DeepDyve Pubget Overpricing