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10.1101/cshperspect.a015545 http://scihub22266oqcxt.onion/10.1101/cshperspect.a015545 C3662356!3662356!23732858     free     free     free       Cold+Spring+Harb+Perspect+Med 2013 ; 3 (6): ä Nephropedia Template TP {{tp|p=23732858|t=2013. Regulatory Cells and Transplantation Tolerance |pdf=|usr=}}{{23732858}} gab.com Text Regulatory Cells and Transplantation Tolerance JO: Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=23732858 #FOAvip Transplantation tolerance is a continuing therapeutic goal, and it is now clear that a subpopulation of T cells with regulatory activity (Treg) that express the transcription factor foxp3 are crucial to this aspiration. Although reprogramming of the immune system to donor-specific transplantation tolerance can be readily achieved in adult mouse models, it has yet to be successfully translated in human clinical practice. This requires that we understand the fundamental mechanisms by which donor antigen-specific Treg are induced and function to maintain tolerance, so that we can target therapies to enhance rather than impede these regulatory processes. Our current understanding is that Treg act via numerous molecular mechanisms, and critical underlying components such as mTOR inhibition, are only now emerging. Twit Text FOAVip Regulatory Cells and Transplantation Tolerance ????Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=23732858 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23732858 #FOAvip English Wikipedia <ref>{{Cite pmid|23732858}}</ref> Regulatory Cells and Transplantation Tolerance #MMPMID23732858 Cobbold SP; Waldmann H Cold Spring Harb Perspect Med 2013[Jun]; 3 (6): ä PMID23732858show ga Transplantation tolerance is a continuing therapeutic goal, and it is now clear that a subpopulation of T cells with regulatory activity (Treg) that express the transcription factor foxp3 are crucial to this aspiration. Although reprogramming of the immune system to donor-specific transplantation tolerance can be readily achieved in adult mouse models, it has yet to be successfully translated in human clinical practice. This requires that we understand the fundamental mechanisms by which donor antigen-specific Treg are induced and function to maintain tolerance, so that we can target therapies to enhance rather than impede these regulatory processes. Our current understanding is that Treg act via numerous molecular mechanisms, and critical underlying components such as mTOR inhibition, are only now emerging. äRegulatory Cells and Transplantation Tolerance (epmc).pdf DeepDyve Pubget Overpricing