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10.1007/s11897-013-0133-0 http://scihub22266oqcxt.onion/10.1007/s11897-013-0133-0 C3647019!3647019!23508767     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Heart+Fail+Rep 2013 ; 10 (2): 109-21 Nephropedia Template TP {{tp|p=23508767|t=2013. Cardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications |pdf=|usr=}}{{23508767}} gab.com Text Cardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications JO: Curr Heart Fail Rep http://www.kidney.de/pget.php?&tw=1&pmid=23508767 #FOAvip Diabetes and obesity are both associated with lipotoxic cardiomyopathy exclusive of coronary artery disease and hypertension. Lipotoxicities have become a public health concern and are responsible for a significant portion of clinical cardiac disease. These abnormalities may be the result of a toxic metabolic shift to more fatty acid and less glucose oxidation with concomitant accumulation of toxic lipids. Lipids can directly alter cellular structures and activate downstream pathways leading to toxicity. Recent data have implicated fatty acids and fatty acyl coenzyme A, diacylglycerol and ceramide in cellular lipotoxicity, which may be caused by apoptosis, defective insulin signaling, endoplasmic reticulum stress, activation of protein kinase C, MAPK activation or modulation of PPARs. Twit Text FOAVip Cardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications ????Curr Heart Fail Rep http://www.kidney.de/pget.php?&tw=1&pmid=23508767 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23508767 #FOAvip English Wikipedia <ref>{{Cite pmid|23508767}}</ref> Cardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications #MMPMID23508767 Drosatos K; Schulze PC Curr Heart Fail Rep 2013[Jun]; 10 (2): 109-21 PMID23508767show ga Diabetes and obesity are both associated with lipotoxic cardiomyopathy exclusive of coronary artery disease and hypertension. Lipotoxicities have become a public health concern and are responsible for a significant portion of clinical cardiac disease. These abnormalities may be the result of a toxic metabolic shift to more fatty acid and less glucose oxidation with concomitant accumulation of toxic lipids. Lipids can directly alter cellular structures and activate downstream pathways leading to toxicity. Recent data have implicated fatty acids and fatty acyl coenzyme A, diacylglycerol and ceramide in cellular lipotoxicity, which may be caused by apoptosis, defective insulin signaling, endoplasmic reticulum stress, activation of protein kinase C, MAPK activation or modulation of PPARs. äCardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications (epmc).pdf DeepDyve Pubget Overpricing