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10.1099/vir.0.030346-0 http://scihub22266oqcxt.onion/10.1099/vir.0.030346-0 C3168279!3168279 !21367984     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=21367984 &cmd=llinks): Failed to open stream: HTTP request failed! 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Cytokine production by primary human macrophages infected with highly pathogenic H5N1 or pandemic H1N1 2009 influenza viruses |pdf=|usr=}}{{21367984 }} gab.com Text Cytokine production by primary human macrophages infected with highly pathogenic H5N1 or pandemic H1N1 2009 influenza viruses JO: J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=21367984 #FOAvip Highly pathogenic H5N1 avian influenza viruses have caused infection in humans, with a high mortality rate, since 1997. While the pathogenesis of this infection is not completely understood, hypercytokinaemia and alveolar macrophages are thought to play a role. To gain further insight into the cytokine-mediated pathogenesis of this infection in humans, we measured various cytokines produced by primary human macrophages infected with H5N1, pandemic H1N1 or seasonal influenza viruses. We found that many cytokines were produced at higher levels on infection with the H5N1 strains tested compared with seasonal influenza viruses. Interestingly, the extent of cytokine induction varied among the H5N1 strains and did not correlate with replicative ability in macrophages. Further, a pandemic H1N1 virus induced higher levels of several cytokines compared with seasonal viruses and some H5N1 strains. Our results demonstrate that high cytokine induction is not a universal feature of all H5N1 viruses. Twit Text FOAVip Cytokine production by primary human macrophages infected with highly pathogenic H5N1 or pandemic H1N1 2009 influenza viruses ????J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=21367984 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=21367984 #FOAvip English Wikipedia <ref>{{Cite pmid|21367984 }}</ref> Cytokine production by primary human macrophages infected with highly pathogenic H5N1 or pandemic H1N1 2009 influenza viruses #MMPMID21367984 Sakabe S ; Iwatsuki-Horimoto K ; Takano R ; Nidom CA ; Le MTQ ; Nagamura-Inoue T ; Horimoto T ; Yamashita N ; Kawaoka Y J Gen Virol 2011[Jun]; 92 (Pt 6 ): 1428-1434 PMID21367984 show ga Highly pathogenic H5N1 avian influenza viruses have caused infection in humans, with a high mortality rate, since 1997. While the pathogenesis of this infection is not completely understood, hypercytokinaemia and alveolar macrophages are thought to play a role. To gain further insight into the cytokine-mediated pathogenesis of this infection in humans, we measured various cytokines produced by primary human macrophages infected with H5N1, pandemic H1N1 or seasonal influenza viruses. We found that many cytokines were produced at higher levels on infection with the H5N1 strains tested compared with seasonal influenza viruses. Interestingly, the extent of cytokine induction varied among the H5N1 strains and did not correlate with replicative ability in macrophages. Further, a pandemic H1N1 virus induced higher levels of several cytokines compared with seasonal viruses and some H5N1 strains. Our results demonstrate that high cytokine induction is not a universal feature of all H5N1 viruses. |*Pandemics [MESH] |Cells, Cultured [MESH] |Cytokines/genetics/*immunology [MESH] |Gene Expression [MESH] |Humans [MESH] |Influenza A Virus, H1N1 Subtype/immunology/*physiology [MESH] |Influenza A Virus, H5N1 Subtype/immunology/*physiology [MESH] |Influenza, Human/epidemiology/*immunology/virology [MESH] |Japan [MESH]Cytokine production by primary human macrophages infected with highly (epmc).pdf DeepDyve Pubget Overpricing