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10.1093/infdis/jiq018 http://scihub22266oqcxt.onion/10.1093/infdis/jiq018 C3086437!3086437 !21148495     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=21148495 &cmd=llinks): Failed to open stream: HTTP request failed! 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Infection with highly pathogenic H7 influenza viruses results in an attenuated proinflammatory cytokine and chemokine response early after infection |pdf=|usr=}}{{21148495 }} gab.com Text Infection with highly pathogenic H7 influenza viruses results in an attenuated proinflammatory cytokine and chemokine response early after infection JO: J Infect Dis http://www.kidney.de/pget.php?&tw=1&pmid=21148495 #FOAvip Avian influenza A viruses of the H7 subtype have resulted in more than 100 cases of human infection since 2002. Highly pathogenic avian influenza (HPAI) H7 viruses have the capacity to cause severe respiratory disease and even death; however, the induction of the human innate immune response to H7 virus infection has not been well characterized. To better understand H7 virus pathogenesis in the human respiratory tract, we employed a polarized human bronchial epithelial cell model and primary human monocyte-derived macrophages. Here, we show that infection with HPAI H7 viruses resulted in a delayed and weakened production of cytokines, including the type I interferon response, compared with infections of other influenza A subtypes, including H7 viruses of low pathogenicity. These studies revealed that H7 viruses vary greatly in their ability to activate host innate responses and may contribute to the virulence of these viruses observed in humans. Twit Text FOAVip Infection with highly pathogenic H7 influenza viruses results in an attenuated proinflammatory cytokine and chemokine response early after infection ????J Infect Dis http://www.kidney.de/pget.php?&tw=1&pmid=21148495 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=21148495 #FOAvip English Wikipedia <ref>{{Cite pmid|21148495 }}</ref> Infection with highly pathogenic H7 influenza viruses results in an attenuated proinflammatory cytokine and chemokine response early after infection #MMPMID21148495 Belser JA ; Zeng H ; Katz JM ; Tumpey TM J Infect Dis 2011[Jan]; 203 (1 ): 40-8 PMID21148495 show ga Avian influenza A viruses of the H7 subtype have resulted in more than 100 cases of human infection since 2002. Highly pathogenic avian influenza (HPAI) H7 viruses have the capacity to cause severe respiratory disease and even death; however, the induction of the human innate immune response to H7 virus infection has not been well characterized. To better understand H7 virus pathogenesis in the human respiratory tract, we employed a polarized human bronchial epithelial cell model and primary human monocyte-derived macrophages. Here, we show that infection with HPAI H7 viruses resulted in a delayed and weakened production of cytokines, including the type I interferon response, compared with infections of other influenza A subtypes, including H7 viruses of low pathogenicity. These studies revealed that H7 viruses vary greatly in their ability to activate host innate responses and may contribute to the virulence of these viruses observed in humans. |Adolescent [MESH] |Adult [MESH] |Cell Death [MESH] |Cells, Cultured [MESH] |Cytokines/*biosynthesis/*immunology [MESH] |Epithelial Cells/*immunology/virology [MESH] |Gene Expression Profiling [MESH] |Humans [MESH] |Influenza A virus/*immunology/pathogenicity [MESH] |Influenza, Human/*immunology/*virology [MESH] |Macrophages/*immunology/virology [MESH] |RNA, Messenger/biosynthesis/genetics [MESH] |Reverse Transcriptase Polymerase Chain Reaction [MESH] |Virulence [MESH]Infection with highly pathogenic H7 influenza viruses results in an at(epmc).pdf DeepDyve Pubget Overpricing