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10.1161/STROKEAHA.109.558346 http://scihub22266oqcxt.onion/10.1161/STROKEAHA.109.558346 C2774207!2774207 !19644062     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=19644062 &cmd=llinks): Failed to open stream: HTTP request failed! 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Gene variation of the transient receptor potential cation channel, subfamily M, member 7 (TRPM7), and risk of incident ischemic stroke: prospective, nested, case-control study |pdf=|usr=}}{{19644062 }} gab.com Text Gene variation of the transient receptor potential cation channel, subfamily M, member 7 (TRPM7), and risk of incident ischemic stroke: prospective, nested, case-control study JO: Stroke http://www.kidney.de/pget.php?&tw=1&pmid=19644062 #FOAvip BACKGROUND AND PURPOSE: Transient receptor potential cation channel, subfamily M, member 7 (TRPM7), has been implicated in ischemic brain damage, a major source of morbidity and mortality in westernized society. We hypothesized that TRPM7 gene variation might play a role in the risk of ischemic stroke. METHODS: From a group of DNA samples collected at baseline in a prospective cohort of 14 916 initially healthy American men, we assessed 16 TRPM7 tag-single-nucleotide polymorphisms (SNPs) (dbSNP: rs11854949, rs4775899, rs11635825, rs12905120, rs16973487, rs7173321, rs7163283, rs17520378, rs17520350, rs4775892, rs7174839, rs17645523, rs3109894, rs616256, rs11070795, and rs313158) from 245 white men who subsequently had an incident ischemic stroke and from 245 age- and smoking habit-matched white men who remained free of reported vascular disease during follow-up (controls). RESULTS: All SNPs examined were in Hardy-Weinberg equilibrium. Overall allele, genotype, and haplotype distributions were similar between cases and controls. Marker-by-marker conditional logistic-regression analysis, adjusted for potential risk factors, showed no evidence for an association between any of the SNPs tested and ischemic stroke. Further investigation with an Entropy Blocker-defined, haplotype-based approach showed similar null findings. Prespecified analysis limited to participants without baseline diabetes and hypertension (ie, low-risk group) again showed similar null findings. CONCLUSIONS: The present prospective investigation provides no evidence of a role for the TRPM7 gene in the risk of incident ischemic stroke. Twit Text FOAVip Gene variation of the transient receptor potential cation channel, subfamily M, member 7 (TRPM7), and risk of incident ischemic stroke: prospective, nested, case-control study ????Stroke http://www.kidney.de/pget.php?&tw=1&pmid=19644062 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19644062 #FOAvip English Wikipedia <ref>{{Cite pmid|19644062 }}</ref> Gene variation of the transient receptor potential cation channel, subfamily M, member 7 (TRPM7), and risk of incident ischemic stroke: prospective, nested, case-control study #MMPMID19644062 Romero JR ; Ridker PM ; Zee RY Stroke 2009[Sep]; 40 (9 ): 2965-8 PMID19644062 show ga BACKGROUND AND PURPOSE: Transient receptor potential cation channel, subfamily M, member 7 (TRPM7), has been implicated in ischemic brain damage, a major source of morbidity and mortality in westernized society. We hypothesized that TRPM7 gene variation might play a role in the risk of ischemic stroke. METHODS: From a group of DNA samples collected at baseline in a prospective cohort of 14 916 initially healthy American men, we assessed 16 TRPM7 tag-single-nucleotide polymorphisms (SNPs) (dbSNP: rs11854949, rs4775899, rs11635825, rs12905120, rs16973487, rs7173321, rs7163283, rs17520378, rs17520350, rs4775892, rs7174839, rs17645523, rs3109894, rs616256, rs11070795, and rs313158) from 245 white men who subsequently had an incident ischemic stroke and from 245 age- and smoking habit-matched white men who remained free of reported vascular disease during follow-up (controls). RESULTS: All SNPs examined were in Hardy-Weinberg equilibrium. Overall allele, genotype, and haplotype distributions were similar between cases and controls. Marker-by-marker conditional logistic-regression analysis, adjusted for potential risk factors, showed no evidence for an association between any of the SNPs tested and ischemic stroke. Further investigation with an Entropy Blocker-defined, haplotype-based approach showed similar null findings. Prespecified analysis limited to participants without baseline diabetes and hypertension (ie, low-risk group) again showed similar null findings. CONCLUSIONS: The present prospective investigation provides no evidence of a role for the TRPM7 gene in the risk of incident ischemic stroke. |*Polymorphism, Single Nucleotide [MESH] |Adult [MESH] |Aged [MESH] |Aged, 80 and over [MESH] |Brain Ischemia/*genetics [MESH] |Female [MESH] |Humans [MESH] |Male [MESH] |Middle Aged [MESH] |Prospective Studies [MESH] |Protein Serine-Threonine Kinases [MESH] |Risk Factors [MESH] |Stroke/*genetics [MESH]Gene variation of the transient receptor potential cation channel, sub(epmc).pdf DeepDyve Pubget Overpricing