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http://scihub22266oqcxt.onion/ C2736785!2736785!17348295     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Comp+Med 2007 ; 57 (1): 82-9 Nephropedia Template TP {{tp|p=17348295|t=2007. Induction of Pro- and Anti-inflammatory Molecules in a Mouse Model of Pneumococcal Pneumonia after Influenza |pdf=|usr=}}{{17348295}} gab.com Text Induction of Pro- and Anti-inflammatory Molecules in a Mouse Model of Pneumococcal Pneumonia after Influenza JO: Comp Med http://www.kidney.de/pget.php?&tw=1&pmid=17348295 #FOAvip Mortality after influenza is often due to secondary bacterial pneumonia with Streptococcus pneumoniae, particularly in the elderly. The reasons for the high fatality rate seen with this disease are unclear. To further characterize the pathogenesis of pneumonia after influenza in a mouse model, we examined the pathology and immunology that leads to fatal infection. Influenza-infected mice were either euthanized 24 h after secondary infection with S. pneumoniae for determination of pathology, bacterial cultures, and levels of immune effectors or were followed by use of a live imaging system for development of pneumonia. Influenza-infected mice challenged with each of 3 serotypes of pneumococcus developed a severe, necrotic pneumonia and met endpoints for euthanasia in 24 to 60 h. Strikingly elevated levels of both pro- and anti-inflammatory molecules including interleukins 6 and 10, macrophage inflammatory protein 1?, and chemokine KC were present in the blood. High levels of these cytokines and chemokines as well as tumor necrosis factor ?, interleukin 1?, and heme oxygenase 1 were present in the lungs, accompanied by a massive influx of neutrophils. Mortality correlated with the development of pneumonia and lung inflammation but not with bacteremia. This model has the potential to help us understand the pathogenesis of severe lung infections. Twit Text FOAVip Induction of Pro- and Anti-inflammatory Molecules in a Mouse Model of Pneumococcal Pneumonia after Influenza ????Comp Med http://www.kidney.de/pget.php?&tw=1&pmid=17348295 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=17348295 #FOAvip English Wikipedia <ref>{{Cite pmid|17348295}}</ref> Induction of Pro- and Anti-inflammatory Molecules in a Mouse Model of Pneumococcal Pneumonia after Influenza #MMPMID17348295 Smith MW; Schmidt JE; Rehg JE; Orihuela CJ; McCullers JA Comp Med 2007[Feb]; 57 (1): 82-9 PMID17348295show ga Mortality after influenza is often due to secondary bacterial pneumonia with Streptococcus pneumoniae, particularly in the elderly. The reasons for the high fatality rate seen with this disease are unclear. To further characterize the pathogenesis of pneumonia after influenza in a mouse model, we examined the pathology and immunology that leads to fatal infection. Influenza-infected mice were either euthanized 24 h after secondary infection with S. pneumoniae for determination of pathology, bacterial cultures, and levels of immune effectors or were followed by use of a live imaging system for development of pneumonia. Influenza-infected mice challenged with each of 3 serotypes of pneumococcus developed a severe, necrotic pneumonia and met endpoints for euthanasia in 24 to 60 h. Strikingly elevated levels of both pro- and anti-inflammatory molecules including interleukins 6 and 10, macrophage inflammatory protein 1?, and chemokine KC were present in the blood. High levels of these cytokines and chemokines as well as tumor necrosis factor ?, interleukin 1?, and heme oxygenase 1 were present in the lungs, accompanied by a massive influx of neutrophils. Mortality correlated with the development of pneumonia and lung inflammation but not with bacteremia. This model has the potential to help us understand the pathogenesis of severe lung infections. äInduction of Pro- and Anti-inflammatory Molecules in a Mouse Model of (epmc).pdf DeepDyve Pubget Overpricing