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10.1046/j.1523-1755.1998.00196.x http://scihub22266oqcxt.onion/10.1046/j.1523-1755.1998.00196.x 9853247!ä!9853247 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Kidney+Int 1998 ; 54 (6): 1832-42 Nephropedia Template TP {{tp|p=9853247|t=1998. Architectural patterns in branching morphogenesis in the kidney |pdf=|usr=}}{{9853247}} gab.com Text Architectural patterns in branching morphogenesis in the kidney JO: Kidney Int http://www.kidney.de/pget.php?&tw=1&pmid=9853247 #FOAvip During kidney development, several discrete steps generate its three-dimensional pattern including specific branch types, regional differential growth of stems, the specific axes of growth and temporal progression of the pattern. The ureteric bud undergoes three different types of branching. In the first, terminal bifid type, a lateral branch arises and immediately bifurcates to form two terminal branches whose tips induce the formation of nephrons. After 15 such divisions (in humans) of this specifically renal type of branching, several nephrons are induced whose connecting tubules fuse and elongate to form the arcades. Finally, the last generations undergo strictly lateral branching to form the cortical system. The stems of these branches elongate in a highly regulated pattern. The molecular basis of these processes is unknown and we briefly review their potential mediators. Differential growth in three different axes of the kidney (cortico-medullary, dorsoventral and rostro-caudal) generate the characteristic shape of the kidney. Rapid advances in molecular genetics highlight the need for development of specific assays for each of these discrete steps, a prerequisite for identification of the involved pathways. The identification of molecules that control branching (the ultimate determinant of the number of nephrons) has acquired new urgency with the recent suggestion that a reduced nephron number predisposes humans to hypertension and to progression of renal failure. Twit Text FOAVip Architectural patterns in branching morphogenesis in the kidney ????Kidney Int http://www.kidney.de/pget.php?&tw=1&pmid=9853247 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=9853247 #FOAvip English Wikipedia <ref>{{Cite pmid|9853247}}</ref> Architectural patterns in branching morphogenesis in the kidney #MMPMID9853247 al-Awqati Q; Goldberg MR Kidney Int 1998[Dec]; 54 (6): 1832-42 PMID9853247show ga During kidney development, several discrete steps generate its three-dimensional pattern including specific branch types, regional differential growth of stems, the specific axes of growth and temporal progression of the pattern. The ureteric bud undergoes three different types of branching. In the first, terminal bifid type, a lateral branch arises and immediately bifurcates to form two terminal branches whose tips induce the formation of nephrons. After 15 such divisions (in humans) of this specifically renal type of branching, several nephrons are induced whose connecting tubules fuse and elongate to form the arcades. Finally, the last generations undergo strictly lateral branching to form the cortical system. The stems of these branches elongate in a highly regulated pattern. The molecular basis of these processes is unknown and we briefly review their potential mediators. Differential growth in three different axes of the kidney (cortico-medullary, dorsoventral and rostro-caudal) generate the characteristic shape of the kidney. Rapid advances in molecular genetics highlight the need for development of specific assays for each of these discrete steps, a prerequisite for identification of the involved pathways. The identification of molecules that control branching (the ultimate determinant of the number of nephrons) has acquired new urgency with the recent suggestion that a reduced nephron number predisposes humans to hypertension and to progression of renal failure. |Animals[MESH] |Drosophila/embryology[MESH] |Embryo, Nonmammalian/physiology[MESH] |Embryonic and Fetal Development/physiology[MESH] |Humans[MESH] |Kidney/*embryology[MESH] |Nephrons/embryology[MESH]Architectural patterns in branching morphogenesis in the kidney (epmc).pdf DeepDyve Pubget Overpricing