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10.3109/13550289509111016 http://scihub22266oqcxt.onion/10.3109/13550289509111016 9222348!ä!9222348 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Neurovirol 1995 ; 1 (1): 118-25 Nephropedia Template TP {{tp|p=9222348|t=1995. Effect of neurotropic virus infection on neuronal and inducible nitric oxide synthase activity in rat brain |pdf=|usr=}}{{9222348}} gab.com Text Effect of neurotropic virus infection on neuronal and inducible nitric oxide synthase activity in rat brain JO: J Neurovirol http://www.kidney.de/pget.php?&tw=1&pmid=9222348 #FOAvip To elucidate the potential role of inducible nitric oxide synthase (iNOS) and neuronal constitutive nitric oxide synthase (cNOS) in the pathogenesis of virus-induced encephalopathy, the activities of both NOS isoforms were determined in the brains of rats infected with Borna disease virus (BDV) or rabies virus. iNOS activity strongly increased, whereas neuronal cNOS activity significantly decreased in a time-dependent manner after either BDV or rabies virus infection. Choline acetyltransferase activity in the brain remained unchanged during both virus infections, suggesting that the decrease in cNOS activity does not reflect a generalized neuronal loss. Immunohistochemistry and Northern blot analyses indicate that the decrease in neuronal cNOS activity is due to a decrease in cNOS protein and mRNA synthesis. These results suggest that both an excessive generation of NO by activated macrophages or microglia, as well as a decrease of NO production in neurons may contribute to the neuropathogenesis of neurotropic virus infections. Twit Text FOAVip Effect of neurotropic virus infection on neuronal and inducible nitric oxide synthase activity in rat brain ????J Neurovirol http://www.kidney.de/pget.php?&tw=1&pmid=9222348 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=9222348 #FOAvip English Wikipedia <ref>{{Cite pmid|9222348}}</ref> Effect of neurotropic virus infection on neuronal and inducible nitric oxide synthase activity in rat brain #MMPMID9222348 Akaike T; Weihe E; Schaefer M; Fu ZF; Zheng YM; Vogel W; Schmidt H; Koprowski H; Dietzschold B J Neurovirol 1995[Mar]; 1 (1): 118-25 PMID9222348show ga To elucidate the potential role of inducible nitric oxide synthase (iNOS) and neuronal constitutive nitric oxide synthase (cNOS) in the pathogenesis of virus-induced encephalopathy, the activities of both NOS isoforms were determined in the brains of rats infected with Borna disease virus (BDV) or rabies virus. iNOS activity strongly increased, whereas neuronal cNOS activity significantly decreased in a time-dependent manner after either BDV or rabies virus infection. Choline acetyltransferase activity in the brain remained unchanged during both virus infections, suggesting that the decrease in cNOS activity does not reflect a generalized neuronal loss. Immunohistochemistry and Northern blot analyses indicate that the decrease in neuronal cNOS activity is due to a decrease in cNOS protein and mRNA synthesis. These results suggest that both an excessive generation of NO by activated macrophages or microglia, as well as a decrease of NO production in neurons may contribute to the neuropathogenesis of neurotropic virus infections. |Animals[MESH] |Blotting, Northern[MESH] |Borna Disease/enzymology/*virology[MESH] |Borna disease virus/*enzymology[MESH] |Brain/enzymology/virology[MESH] |Choline O-Acetyltransferase/metabolism[MESH] |Encephalitis, Viral/enzymology/virology[MESH] |Female[MESH] |Immunohistochemistry[MESH] |Kinetics[MESH] |Nitric Oxide Synthase/genetics/*metabolism[MESH] |RNA, Messenger/analysis[MESH] |Rabies virus/*enzymology[MESH] |Rabies/enzymology/*virology[MESH] |Rats[MESH]Effect of neurotropic virus infection on neuronal and inducible nitric(epmc).pdf DeepDyve Pubget Overpricing