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10.1016/0006-3223(95)00480-7 http://scihub22266oqcxt.onion/10.1016/0006-3223(95)00480-7 8886296!ä!8886296 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biol+Psychiatry 1996 ; 40 (7): 629-36 Nephropedia Template TP {{tp|p=8886296|t=1996. Prefrontal cortex dysfunction in Borna disease virus (BDV)--infected rats |pdf=|usr=}}{{8886296}} gab.com Text Prefrontal cortex dysfunction in Borna disease virus (BDV)--infected rats JO: Biol Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=8886296 #FOAvip Viruses have been proposed to play a role in the pathogenesis of schizophrenia; however, the mechanisms by which infection could cause the affective, cognitive, and movement disorders of schizophrenia are not understood. The neurotropic RNA virus, Borna disease (BD) virus, linked to schizophrenia by serologic studies, causes movement and behavior disorders in a wide variety of mammalian and bird hosts. BD rats have hyperactivity and stereotyped behaviors similar to those that follow neurotoxic or electrolytic lesions in frontal cortex or its catecholamine afferents in rats. BD rats have high levels of viral nucleic acid in the prefrontal cortex (PFC), abnormal mesocortical dopamine activity (elevated levels of DOPAC in PFC), yet no alteration in specific binding of D1 or D2 receptor radioligands in PFC. Since frontal lobe dysfunction is frequently reported in schizophrenia, the BD rat model may provide insights into pathogenesis and management of this debilitating psychiatric disease. Twit Text FOAVip Prefrontal cortex dysfunction in Borna disease virus (BDV)--infected rats ????Biol Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=8886296 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=8886296 #FOAvip English Wikipedia <ref>{{Cite pmid|8886296}}</ref> Prefrontal cortex dysfunction in Borna disease virus (BDV)--infected rats #MMPMID8886296 Solbrig MV; Koob GF; Fallon JH; Reid S; Lipkin WI Biol Psychiatry 1996[Oct]; 40 (7): 629-36 PMID8886296show ga Viruses have been proposed to play a role in the pathogenesis of schizophrenia; however, the mechanisms by which infection could cause the affective, cognitive, and movement disorders of schizophrenia are not understood. The neurotropic RNA virus, Borna disease (BD) virus, linked to schizophrenia by serologic studies, causes movement and behavior disorders in a wide variety of mammalian and bird hosts. BD rats have hyperactivity and stereotyped behaviors similar to those that follow neurotoxic or electrolytic lesions in frontal cortex or its catecholamine afferents in rats. BD rats have high levels of viral nucleic acid in the prefrontal cortex (PFC), abnormal mesocortical dopamine activity (elevated levels of DOPAC in PFC), yet no alteration in specific binding of D1 or D2 receptor radioligands in PFC. Since frontal lobe dysfunction is frequently reported in schizophrenia, the BD rat model may provide insights into pathogenesis and management of this debilitating psychiatric disease. |3,4-Dihydroxyphenylacetic Acid/metabolism[MESH] |Animals[MESH] |Autoradiography[MESH] |Borna Disease/*physiopathology[MESH] |Borna disease virus/genetics[MESH] |Brain Mapping[MESH] |Dopamine/physiology[MESH] |Gene Expression Regulation, Viral/physiology[MESH] |Male[MESH] |Motor Activity/physiology[MESH] |Neurocognitive Disorders/*physiopathology[MESH] |Prefrontal Cortex/*physiopathology[MESH] |RNA Probes[MESH] |Rats[MESH] |Rats, Inbred Lew[MESH] |Receptors, Dopamine D1/*physiology[MESH] |Receptors, Dopamine D2/*physiology[MESH]Prefrontal cortex dysfunction in Borna disease virus (BDV)--infected r(epmc).pdf DeepDyve Pubget Overpricing