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10.1164/arrd.1977.115.2.269 http://scihub22266oqcxt.onion/10.1164/arrd.1977.115.2.269 842940!ä!842940 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+Rev+Respir+Dis 1977 ; 115 (2): 269-77 Nephropedia Template TP {{tp|p=842940|t=1977. Effect of influenza viral infection on the ingestion and killing of bacteria by alveolar macrophages |pdf=|usr=}}{{842940}} gab.com Text Effect of influenza viral infection on the ingestion and killing of bacteria by alveolar macrophages JO: Am Rev Respir Dis http://www.kidney.de/pget.php?&tw=1&pmid=842940 #FOAvip In experimental animals, influenza prediposes the lung to superinfection by reducing the antibacterial efficiency of the alveolar macrophage system. Because such defects may represent abnormalities in ingestion or inactivation of inhaled bacteria, these subcomponents of phagocytosis were tested in mice infected 5 days previously with influenza A virus (NWS or WSN). The mice were exposed to aerosols of Staphylococcus epidermidis and then the rates of bacterial inactivation and percentages of intracellularly located staphylococci were measured. Rates of bacterial inactivation were determined for the left lung by pour-plate enumeration methods. The percentage of ingested bacteria was determined in the in situ perfused right lung by histologically determining the intra- or extracellular location of 100 or more staphylococci. Rates of inactivation of S. epidermidis at 4 hours after bacterial challenge were: control, 90.1 per cent; WSN, 73.0 per cent; NWS, 68.6 per cent, P less than 0.01. The percentage of intracellular staphylococci at 4 hours were: control, 90.9 per cent; WSN, 69.9 per cent; and NWS, 73.8 per cent, P less than 0.01. Microcolonies of proliferating staphylococci were also observed within macrophages of mice infected with each strain of influenza. These experiments demonstrated that in this experimental model, influenzal infection impairs the inactivation of inhaled bacteria by retarding the ingestion of bacteria and by allowing bacteria to proliferate within macrophages. Twit Text FOAVip Effect of influenza viral infection on the ingestion and killing of bacteria by alveolar macrophages ????Am Rev Respir Dis http://www.kidney.de/pget.php?&tw=1&pmid=842940 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=842940 #FOAvip English Wikipedia <ref>{{Cite pmid|842940}}</ref> Effect of influenza viral infection on the ingestion and killing of bacteria by alveolar macrophages #MMPMID842940 Warshauer D; Goldstein E; Akers T; Lippert W; Kim M Am Rev Respir Dis 1977[Feb]; 115 (2): 269-77 PMID842940show ga In experimental animals, influenza prediposes the lung to superinfection by reducing the antibacterial efficiency of the alveolar macrophage system. Because such defects may represent abnormalities in ingestion or inactivation of inhaled bacteria, these subcomponents of phagocytosis were tested in mice infected 5 days previously with influenza A virus (NWS or WSN). The mice were exposed to aerosols of Staphylococcus epidermidis and then the rates of bacterial inactivation and percentages of intracellularly located staphylococci were measured. Rates of bacterial inactivation were determined for the left lung by pour-plate enumeration methods. The percentage of ingested bacteria was determined in the in situ perfused right lung by histologically determining the intra- or extracellular location of 100 or more staphylococci. Rates of inactivation of S. epidermidis at 4 hours after bacterial challenge were: control, 90.1 per cent; WSN, 73.0 per cent; NWS, 68.6 per cent, P less than 0.01. The percentage of intracellular staphylococci at 4 hours were: control, 90.9 per cent; WSN, 69.9 per cent; and NWS, 73.8 per cent, P less than 0.01. Microcolonies of proliferating staphylococci were also observed within macrophages of mice infected with each strain of influenza. These experiments demonstrated that in this experimental model, influenzal infection impairs the inactivation of inhaled bacteria by retarding the ingestion of bacteria and by allowing bacteria to proliferate within macrophages. |*Bacteriolysis[MESH] |*Staphylococcus[MESH] |Animals[MESH] |Influenza A virus/*pathogenicity[MESH] |Macrophages/*microbiology[MESH] |Mice[MESH] |Orthomyxoviridae Infections/*microbiology[MESH] |Pulmonary Alveoli/*microbiology/pathology[MESH] |Time Factors[MESH]Effect of influenza viral infection on the ingestion and killing of ba(epmc).pdf DeepDyve Pubget Overpricing