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10.1016/0005-2736(93)90023-s http://scihub22266oqcxt.onion/10.1016/0005-2736(93)90023-s 8391320!ä!8391320 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biochim+Biophys+Acta 1993 ; 1149 (1): 49-54 Nephropedia Template TP {{tp|p=8391320|t=1993. Na(+)- and anion-dependent Mg2+ influx in isolated hepatocytes |pdf=|usr=}}{{8391320}} gab.com Text Na(+)- and anion-dependent Mg2+ influx in isolated hepatocytes JO: Biochim Biophys Acta http://www.kidney.de/pget.php?&tw=1&pmid=8391320 #FOAvip Hepatocytes, which were Mg(2+)-depleted during isolation, took up Mg2+ during reincubation. Mg2+ uptake was dependent on the concentration of extracellular Mg2+, Na+, Cl-, bicarbonate and phosphate. Li+ and choline+ did not substitute for extracellular Na+ in Mg2+ influx. Mg2+ influx was maximal when all three anion species were present, and did not occur when these anions were replaced by gluconate. Bicarbonate, phosphate and Cl- could substitute for each other. Mg2+ uptake in hepatocytes was inhibited by p-chloromercuribenzene sulfonate, ouabain, gramicidin D, amiloride and verapamil. The results were explained by the assumption that net Mg2+ influx in hepatocytes is operating via electroneutral Na+, Mg2+/anion cotransport driven by the Na+ gradient. However, electrogenic Mg2+ uptake gated by extracellular Na+ and anions could not be excluded. Twit Text FOAVip Na(+)- and anion-dependent Mg2+ influx in isolated hepatocytes ????Biochim Biophys Acta http://www.kidney.de/pget.php?&tw=1&pmid=8391320 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=8391320 #FOAvip English Wikipedia <ref>{{Cite pmid|8391320}}</ref> Na(+)- and anion-dependent Mg2+ influx in isolated hepatocytes #MMPMID8391320 Gunther T; Hollriegl V Biochim Biophys Acta 1993[Jun]; 1149 (1): 49-54 PMID8391320show ga Hepatocytes, which were Mg(2+)-depleted during isolation, took up Mg2+ during reincubation. Mg2+ uptake was dependent on the concentration of extracellular Mg2+, Na+, Cl-, bicarbonate and phosphate. Li+ and choline+ did not substitute for extracellular Na+ in Mg2+ influx. Mg2+ influx was maximal when all three anion species were present, and did not occur when these anions were replaced by gluconate. Bicarbonate, phosphate and Cl- could substitute for each other. Mg2+ uptake in hepatocytes was inhibited by p-chloromercuribenzene sulfonate, ouabain, gramicidin D, amiloride and verapamil. The results were explained by the assumption that net Mg2+ influx in hepatocytes is operating via electroneutral Na+, Mg2+/anion cotransport driven by the Na+ gradient. However, electrogenic Mg2+ uptake gated by extracellular Na+ and anions could not be excluded. |4-Chloromercuribenzenesulfonate/pharmacology[MESH] |Adenosine Triphosphate/metabolism[MESH] |Animals[MESH] |Bicarbonates/pharmacology[MESH] |Biological Transport/drug effects[MESH] |Chlorides/pharmacology[MESH] |In Vitro Techniques[MESH] |Liver/drug effects/*metabolism[MESH] |Magnesium/*metabolism/pharmacology[MESH] |Perfusion[MESH] |Phosphates/pharmacology[MESH] |Rats[MESH]Na(+)- and anion-dependent Mg2+ influx in isolated hepatocytes (epmc).pdf DeepDyve Pubget Overpricing