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10.1002/clc.4960161105 http://scihub22266oqcxt.onion/10.1002/clc.4960161105 8269653!ä!8269653 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Cardiol 1993 ; 16 (11): 768-74 Nephropedia Template TP {{tp|p=8269653|t=1993. Magnesium and the heart: antiarrhythmic therapy with magnesium |pdf=|usr=}}{{8269653}} gab.com Text Magnesium and the heart: antiarrhythmic therapy with magnesium JO: Clin Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=8269653 #FOAvip Magnesium is an essential transmembrane and intracellular modulator of the electrical activity of cardiac cells. This review provides an up-to-date consideration of the cellular and clinical electrophysiological role of magnesium. This ubiquitous element seems to be important from both the theoretical and clinical point of view, because magnesium salts (MgSO4, MgCl2) administered intravenously are particularly effective in those arrhythmias in which the mechanism involves early or delayed after depolarization-induced triggered activity. The authors share the view that I.V. magnesium is the drug of choice in "torsade de pointes" ventricular tachycardia accompanying acquired long QT/QTU syndrome. It is complementary therapeutic agent in digitalis-induced tachycardias. Further studies are needed to elucidate magnesium's mode of action and efficacy in other types of clinical tachyarrhythmias. Twit Text FOAVip Magnesium and the heart: antiarrhythmic therapy with magnesium ????Clin Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=8269653 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=8269653 #FOAvip English Wikipedia <ref>{{Cite pmid|8269653}}</ref> Magnesium and the heart: antiarrhythmic therapy with magnesium #MMPMID8269653 Fazekas T; Scherlag BJ; Vos M; Wellens HJ; Lazzara R Clin Cardiol 1993[Nov]; 16 (11): 768-74 PMID8269653show ga Magnesium is an essential transmembrane and intracellular modulator of the electrical activity of cardiac cells. This review provides an up-to-date consideration of the cellular and clinical electrophysiological role of magnesium. This ubiquitous element seems to be important from both the theoretical and clinical point of view, because magnesium salts (MgSO4, MgCl2) administered intravenously are particularly effective in those arrhythmias in which the mechanism involves early or delayed after depolarization-induced triggered activity. The authors share the view that I.V. magnesium is the drug of choice in "torsade de pointes" ventricular tachycardia accompanying acquired long QT/QTU syndrome. It is complementary therapeutic agent in digitalis-induced tachycardias. Further studies are needed to elucidate magnesium's mode of action and efficacy in other types of clinical tachyarrhythmias. |Animals[MESH] |Arrhythmias, Cardiac/*drug therapy/physiopathology[MESH] |Heart Conduction System/drug effects[MESH] |Heart/*physiology/physiopathology[MESH] |Humans[MESH] |Magnesium Chloride/therapeutic use[MESH] |Magnesium Compounds/pharmacology/*therapeutic use[MESH] |Magnesium Sulfate/therapeutic use[MESH]Magnesium and the heart: antiarrhythmic therapy with magnesium (epmc).pdf DeepDyve Pubget Overpricing