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http://scihub22266oqcxt.onion/ 793408!2032571!793408     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Pathol 1976 ; 85 (2): 277-302 Nephropedia Template TP {{tp|p=793408|t=1976. Spontaneous glomerular sclerosis in aging Sprague-Dawley rats I Lesions associated with mesangial IgM deposits |pdf=|usr=}}{{793408}} gab.com Text Spontaneous glomerular sclerosis in aging Sprague-Dawley rats I Lesions associated with mesangial IgM deposits JO: Am J Pathol http://www.kidney.de/pget.php?&tw=1&pmid=793408 #FOAvip The present studies examined the pathogenesis of focal glomerular sclerosis in aging rats. A marked difference in development of the lesion was noted between males and females, and strain variability was an important factor. Increased glomerular basement membrane permeability with loss of selectivity unrelated to changes in glomerular sialoprotein occurred with aging and was accompanied by increasing proteinuria. Noncomplement-fixing mesangial deposits of rat IgM were present after 1 month of age and were also found in lesser amounts in germfree rats. Fluoresceinated eluates of rat kidneys did not have antibody activity against rat serum or tissue antigens. There was no evidence for a pathogenetic role of IgM deposits. Rat IgG, IgA, IgE, C3, and fibrin were occasionally found in sclerotic areas. Analysis of multiple histologic sections revealed a close correlation between aging and glomerular pathology, with a poor correlation between tubular damage and aging. Glomerular damage appeared to be the initial event leading to tubular damage. Indirect evidence suggests that a relative thymic deficiency may play an important role in the pathogenesis of the lesion. Twit Text FOAVip Spontaneous glomerular sclerosis in aging Sprague-Dawley rats I Lesions associated with mesangial IgM deposits ????Am J Pathol http://www.kidney.de/pget.php?&tw=1&pmid=793408 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=793408 #FOAvip English Wikipedia <ref>{{Cite pmid|793408}}</ref> Spontaneous glomerular sclerosis in aging Sprague-Dawley rats I Lesions associated with mesangial IgM deposits #MMPMID793408 Bolton WK; Benton FR; Maclay JG; Sturgill BC Am J Pathol 1976[Nov]; 85 (2): 277-302 PMID793408show ga The present studies examined the pathogenesis of focal glomerular sclerosis in aging rats. A marked difference in development of the lesion was noted between males and females, and strain variability was an important factor. Increased glomerular basement membrane permeability with loss of selectivity unrelated to changes in glomerular sialoprotein occurred with aging and was accompanied by increasing proteinuria. Noncomplement-fixing mesangial deposits of rat IgM were present after 1 month of age and were also found in lesser amounts in germfree rats. Fluoresceinated eluates of rat kidneys did not have antibody activity against rat serum or tissue antigens. There was no evidence for a pathogenetic role of IgM deposits. Rat IgG, IgA, IgE, C3, and fibrin were occasionally found in sclerotic areas. Analysis of multiple histologic sections revealed a close correlation between aging and glomerular pathology, with a poor correlation between tubular damage and aging. Glomerular damage appeared to be the initial event leading to tubular damage. Indirect evidence suggests that a relative thymic deficiency may play an important role in the pathogenesis of the lesion. |*Aging[MESH] |*Kidney Glomerulus/physiopathology[MESH] |Animals[MESH] |Animals, Newborn[MESH] |Complement Fixation Tests[MESH] |Female[MESH] |Immunoglobulin A[MESH] |Immunoglobulin E[MESH] |Immunoglobulin G[MESH] |Immunoglobulin M/*metabolism[MESH] |Male[MESH] |Nephrosclerosis/etiology/physiopathology/*veterinary[MESH] |Proteinuria[MESH] |Rats[MESH]Spontaneous glomerular sclerosis in aging Sprague-Dawley rats I Lesi(epmc).pdf DeepDyve Pubget Overpricing