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http://scihub22266oqcxt.onion/ 6871495!?!6871495 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=6871495&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Bull+Eur+Physiopathol+Respir 1983 ; 19 (2): 173-8 Nephropedia Template TP {{tp|p=6871495|t=1983. The participation of antiviral immune mechanisms in alveolar macrophage dysfunction during viral pneumonia |pdf=|usr=}}{{6871495}} gab.com Text The participation of antiviral immune mechanisms in alveolar macrophage dysfunction during viral pneumonia JO: Bull Eur Physiopathol Respir http://www.kidney.de/pget.php?&tw=1&pmid=6871495 #FOAvip Virus infections transiently suppress pulmonary antibacterial defenses by causing dysfunctions in the alveolar macrophage phagocytic system. This impairment of pulmonary bactericidal activity is not associated in time with virus proliferation, but rather with the period of time of rapidly declining virus titers and the expression of the antiviral immune response in the lungs. This temporal relationship suggests that the impairment of pulmonary bactericidal activity might be secondary to the antiviral immune response rather than a direct effect of virus replication. Immune depletion of mice during the course of influenza virus pneumonia ameliorated the virus-induced bactericidal defect and prevented bacterial multiplication in the lungs. In contrast, immune reconstitution reestablished the alveolar macrophage phagocytic defect. These data indicate that virus-induced suppression of pulmonary antibacterial defenses may be, in part, immunologically mediated. Twit Text FOAVip The participation of antiviral immune mechanisms in alveolar macrophage dysfunction during viral pneumonia ????Bull Eur Physiopathol Respir http://www.kidney.de/pget.php?&tw=1&pmid=6871495 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=6871495 #FOAvip English Wikipedia <ref>{{Cite pmid|6871495}}</ref> The participation of antiviral immune mechanisms in alveolar macrophage dysfunction during viral pneumonia #MMPMID6871495 Jakab GJ; Warr GA Bull Eur Physiopathol Respir 1983[Mar]; 19 (2): 173-8 PMID6871495show ga Virus infections transiently suppress pulmonary antibacterial defenses by causing dysfunctions in the alveolar macrophage phagocytic system. This impairment of pulmonary bactericidal activity is not associated in time with virus proliferation, but rather with the period of time of rapidly declining virus titers and the expression of the antiviral immune response in the lungs. This temporal relationship suggests that the impairment of pulmonary bactericidal activity might be secondary to the antiviral immune response rather than a direct effect of virus replication. Immune depletion of mice during the course of influenza virus pneumonia ameliorated the virus-induced bactericidal defect and prevented bacterial multiplication in the lungs. In contrast, immune reconstitution reestablished the alveolar macrophage phagocytic defect. These data indicate that virus-induced suppression of pulmonary antibacterial defenses may be, in part, immunologically mediated. |Animals[MESH] |Antilymphocyte Serum/pharmacology[MESH] |Bacterial Infections/*immunology[MESH] |Cyclophosphamide/pharmacology[MESH] |Immunosuppression Therapy[MESH] |In Vitro Techniques[MESH] |Macrophages/*immunology[MESH] |Mice[MESH] |Phagocyte Bactericidal Dysfunction/*immunology[MESH] |Pneumonia, Viral/*immunology[MESH] |Pneumonia/immunology[MESH] |Pulmonary Alveoli/cytology[MESH]The participation of antiviral immune mechanisms in alveolar macrophag(epmc).pdf DeepDyve Pubget Overpricing