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10.1111/j.1600-0773.1984.tb03634.x http://scihub22266oqcxt.onion/10.1111/j.1600-0773.1984.tb03634.x 6324542!ä!6324542 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Acta+Pharmacol+Toxicol+(Copenh) 1984 ; 54 Suppl 1 (ä): 59-65 Nephropedia Template TP {{tp|p=6324542|t=1984. Problems with potassium and magnesium in diuretic-treated patients |pdf=|usr=}}{{6324542}} gab.com Text Problems with potassium and magnesium in diuretic-treated patients JO: Acta Pharmacol Toxicol (Copenh) http://www.kidney.de/pget.php?&tw=1&pmid=6324542 #FOAvip Treatment with thiazides and loop diuretics increase the urinary excretion of potassium and magnesium and the body content of these ions are reduced after long-term treatment. The diuretic-induced magnesium deficiency influences the potassium metabolism. Magnesium is a necessary activator of Na-K-ATPase, which supplies the Na-K pump with energy. Lack of magnesium will therefore impair the pumping of sodium out of the cell and of potassium into the cell. The change of the relationship between extra and intracellular potassium may induce cardiac arrhythmias. Certain groups of patients, such as patients on digitalis therapy, patients with secondary hyperaldosteronism, elderly patients with insufficient dietary habits, and heavy drinkers, run an additional risk of developing potassium/magnesium disturbances. In young patients with uncomplicated essential hypertension, the risk is probably very small. Twit Text FOAVip Problems with potassium and magnesium in diuretic-treated patients ????Acta Pharmacol Toxicol (Copenh) http://www.kidney.de/pget.php?&tw=1&pmid=6324542 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=6324542 #FOAvip English Wikipedia <ref>{{Cite pmid|6324542}}</ref> Problems with potassium and magnesium in diuretic-treated patients #MMPMID6324542 Wester PO; Dyckner T Acta Pharmacol Toxicol (Copenh) 1984[]; 54 Suppl 1 (ä): 59-65 PMID6324542show ga Treatment with thiazides and loop diuretics increase the urinary excretion of potassium and magnesium and the body content of these ions are reduced after long-term treatment. The diuretic-induced magnesium deficiency influences the potassium metabolism. Magnesium is a necessary activator of Na-K-ATPase, which supplies the Na-K pump with energy. Lack of magnesium will therefore impair the pumping of sodium out of the cell and of potassium into the cell. The change of the relationship between extra and intracellular potassium may induce cardiac arrhythmias. Certain groups of patients, such as patients on digitalis therapy, patients with secondary hyperaldosteronism, elderly patients with insufficient dietary habits, and heavy drinkers, run an additional risk of developing potassium/magnesium disturbances. In young patients with uncomplicated essential hypertension, the risk is probably very small. |Diuretics/*adverse effects/therapeutic use[MESH] |Extracellular Space/metabolism[MESH] |Humans[MESH] |Hypertension/drug therapy[MESH] |Magnesium/*metabolism[MESH] |Potassium/blood/*metabolism[MESH]Problems with potassium and magnesium in diuretic-treated patients (epmc).pdf DeepDyve Pubget Overpricing