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http://scihub22266oqcxt.onion/ 4264779!1903922!4264779     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=4264779&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Am+J+Pathol 1973 ; 70 (1): 89-108 Nephropedia Template TP {{tp|p=4264779|t=1973. Plasmodium berghei infection in mice An ultrastructural study of immune complex nephritis |pdf=|usr=}}{{4264779}} gab.com Text Plasmodium berghei infection in mice An ultrastructural study of immune complex nephritis JO: Am J Pathol http://www.kidney.de/pget.php?&tw=1&pmid=4264779 #FOAvip Renal glomeruli of Plasmodium berghei infected mice were studied electron microscopically. The endothelial cell proliferation and endothelial cytoplasmic changes were greatly stimulated on day 7, at the time when there were collections of dense granular aggregates in the glomerular capillary lumens and electron-dense deposits in the mesangial areas. The basement membrane lesion developed with the infiltration of polymorphonuclear leucocytes on day 14 with more electron-dense deposits in the mesangial areas and in the basement membrane. On the basis of these ultrastructural alterations and on previous findings of immune complex (P berghei antigen, mouse globulin and mouse beta1C globulin) deposits by immunofluorescent study, we concluded that glomerular lesions in P berghei infected mice are induced by immune mechanism. Twit Text FOAVip Plasmodium berghei infection in mice An ultrastructural study of immune complex nephritis ????Am J Pathol http://www.kidney.de/pget.php?&tw=1&pmid=4264779 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=4264779 #FOAvip English Wikipedia <ref>{{Cite pmid|4264779}}</ref> Plasmodium berghei infection in mice An ultrastructural study of immune complex nephritis #MMPMID4264779 Boonpucknavig V; Boonpucknavig S; Bhamarapravati N Am J Pathol 1973[Jan]; 70 (1): 89-108 PMID4264779show ga Renal glomeruli of Plasmodium berghei infected mice were studied electron microscopically. The endothelial cell proliferation and endothelial cytoplasmic changes were greatly stimulated on day 7, at the time when there were collections of dense granular aggregates in the glomerular capillary lumens and electron-dense deposits in the mesangial areas. The basement membrane lesion developed with the infiltration of polymorphonuclear leucocytes on day 14 with more electron-dense deposits in the mesangial areas and in the basement membrane. On the basis of these ultrastructural alterations and on previous findings of immune complex (P berghei antigen, mouse globulin and mouse beta1C globulin) deposits by immunofluorescent study, we concluded that glomerular lesions in P berghei infected mice are induced by immune mechanism. |*Plasmodium berghei[MESH] |Animals[MESH] |Antigen-Antibody Complex[MESH] |Female[MESH] |Fluorescent Antibody Technique[MESH] |Immune Complex Diseases/pathology[MESH] |Immune System Diseases/*pathology[MESH] |Kidney Glomerulus/pathology[MESH] |Kidney/pathology[MESH] |Malaria/*immunology[MESH] |Male[MESH] |Mice[MESH] |Microscopy, Electron[MESH]Plasmodium berghei infection in mice An ultrastructural study of immu(epmc).pdf DeepDyve Pubget Overpricing