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10.1007/s10877-025-01393-w http://scihub22266oqcxt.onion/10.1007/s10877-025-01393-w 41369879!?!41369879 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=41369879&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Clin+Monit+Comput 2025 ; ? (?): ? Nephropedia Template TP {{tp|p=41369879|t=2025. Cerebral monitoring responses to bedside physiological challenges in comatose post-cardiac arrest patients |pdf=|usr=}}{{41369879}} gab.com Text Cerebral monitoring responses to bedside physiological challenges in comatose post-cardiac arrest patients JO: J Clin Monit Comput http://www.kidney.de/pget.php?&tw=1&pmid=41369879 #FOAvip To evaluate the effects of three simple bedside challenges on cerebral oxygenation and brain activity, measured non-invasively using near-infrared spectroscopy (NIRS) and frontal single-channel electroencephalography (EEG), in comatose post-cardiac arrest patients, and to examine whether these responses differ according to cerebral autoregulation status and intensive care unit (ICU) outcome and could aid early prognostication. Three bedside physiological challenges were conducted: (1) increasing the fraction of inspired oxygen (FiO(2)) to 100%, (2) lowering the head-of-bed (HOB) to 0 degrees , and (3) elevating end-tidal carbon dioxide (etCO(2)) by 1.0 kPa. Tissue oxygen saturation (StO(2)) and EEG amplitude were hypothesized to increase, by enhancing oxygen delivery (FiO(2)), augmenting cerebral perfusion pressure (HOB), and inducing cerebral vasodilation (etCO(2)). Furthermore, we examined the associations between signal responses, cerebral autoregulation status, and ICU outcome. Of the 48 monitored patients, FiO(2), HOB, and etCO(2) challenges were successfully completed in 41 (85%), 33 (69%), and 32 (67%) patients, respectively. The StO(2) increased on average by 0.3% (95%-CI 0.2-0.5, p < 0.001) for every 10% rise in FiO(2), and 1.94% (95%-CI 0.9-3.0, p < 0.001) for each 15 masculine lowering of the HOB. The etCO(2) challenge did not affect the StO(2). EEG amplitude remained unchanged during all three challenges. No significant differences were found in the responses between patients with intact versus impaired autoregulation or between the ICU outcome groups. Brief physiological challenges simulating common ICU scenarios elicited only modest increases in StO(2), and no measurable response in EEG amplitude. Response patterns were not associated with cerebral autoregulation status or ICU outcome. Twit Text FOAVip Cerebral monitoring responses to bedside physiological challenges in comatose post-cardiac arrest patients ????J Clin Monit Comput http://www.kidney.de/pget.php?&tw=1&pmid=41369879 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=41369879 #FOAvip English Wikipedia <ref>{{Cite pmid|41369879}}</ref> Cerebral monitoring responses to bedside physiological challenges in comatose post-cardiac arrest patients #MMPMID41369879 Kishna YS; Baars S; van der Horst ICC; Mess WH; Hendrix R; Delnoij TSR; Aries M J Clin Monit Comput 2025[Dec]; ? (?): ? PMID41369879show ga To evaluate the effects of three simple bedside challenges on cerebral oxygenation and brain activity, measured non-invasively using near-infrared spectroscopy (NIRS) and frontal single-channel electroencephalography (EEG), in comatose post-cardiac arrest patients, and to examine whether these responses differ according to cerebral autoregulation status and intensive care unit (ICU) outcome and could aid early prognostication. Three bedside physiological challenges were conducted: (1) increasing the fraction of inspired oxygen (FiO(2)) to 100%, (2) lowering the head-of-bed (HOB) to 0 degrees , and (3) elevating end-tidal carbon dioxide (etCO(2)) by 1.0 kPa. Tissue oxygen saturation (StO(2)) and EEG amplitude were hypothesized to increase, by enhancing oxygen delivery (FiO(2)), augmenting cerebral perfusion pressure (HOB), and inducing cerebral vasodilation (etCO(2)). Furthermore, we examined the associations between signal responses, cerebral autoregulation status, and ICU outcome. Of the 48 monitored patients, FiO(2), HOB, and etCO(2) challenges were successfully completed in 41 (85%), 33 (69%), and 32 (67%) patients, respectively. The StO(2) increased on average by 0.3% (95%-CI 0.2-0.5, p < 0.001) for every 10% rise in FiO(2), and 1.94% (95%-CI 0.9-3.0, p < 0.001) for each 15 masculine lowering of the HOB. The etCO(2) challenge did not affect the StO(2). EEG amplitude remained unchanged during all three challenges. No significant differences were found in the responses between patients with intact versus impaired autoregulation or between the ICU outcome groups. Brief physiological challenges simulating common ICU scenarios elicited only modest increases in StO(2), and no measurable response in EEG amplitude. Response patterns were not associated with cerebral autoregulation status or ICU outcome. ?Cerebral monitoring responses to bedside physiological challenges in c(epmc).pdf DeepDyve Pubget Overpricing