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10.3389/fnmol.2023.1239599 http://scihub22266oqcxt.onion/10.3389/fnmol.2023.1239599 37664239!10469682!37664239     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=37664239&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Front+Mol+Neurosci 2023 ; 16 (?): 1239599 Nephropedia Template TP {{tp|p=37664239|t=2023. Ion channels in cancer-induced bone pain: from molecular mechanisms to clinical applications |pdf=|usr=}}{{37664239}} gab.com Text Ion channels in cancer-induced bone pain: from molecular mechanisms to clinical applications JO: Front Mol Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=37664239 #FOAvip Cancer-induced bone pain (CIBP) caused by bone metastasis is one of the most prevalent diseases, and current treatments rely primarily on opioids, which have significant side effects. However, recent developments in pharmaceutical science have identified several new mechanisms for CIBP, including the targeted modification of certain ion channels and receptors. Ion channels are transmembrane proteins, which are situated on biological cell membranes, which facilitate passive transport of inorganic ions across membranes. They are involved in various physiological processes, including transmission of pain signals in the nervous system. In recent years, there has been an increasing interest in the role of ion channels in chronic pain, including CIBP. Therefore, in this review, we summarize the current literature on ion channels, related receptors, and drugs and explore the mechanism of CIBP. Targeting ion channels and regulating their activity might be key to treating pain associated with bone cancer and offer new treatment avenues. Twit Text FOAVip Ion channels in cancer-induced bone pain: from molecular mechanisms to clinical applications ????Front Mol Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=37664239 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=37664239 #FOAvip English Wikipedia <ref>{{Cite pmid|37664239}}</ref> Ion channels in cancer-induced bone pain: from molecular mechanisms to clinical applications #MMPMID37664239 Lu HJ; Wu XB; Wei QQ Front Mol Neurosci 2023[]; 16 (?): 1239599 PMID37664239show ga Cancer-induced bone pain (CIBP) caused by bone metastasis is one of the most prevalent diseases, and current treatments rely primarily on opioids, which have significant side effects. However, recent developments in pharmaceutical science have identified several new mechanisms for CIBP, including the targeted modification of certain ion channels and receptors. Ion channels are transmembrane proteins, which are situated on biological cell membranes, which facilitate passive transport of inorganic ions across membranes. They are involved in various physiological processes, including transmission of pain signals in the nervous system. In recent years, there has been an increasing interest in the role of ion channels in chronic pain, including CIBP. Therefore, in this review, we summarize the current literature on ion channels, related receptors, and drugs and explore the mechanism of CIBP. Targeting ion channels and regulating their activity might be key to treating pain associated with bone cancer and offer new treatment avenues. ?Ion channels in cancer-induced bone pain: from molecular mechanisms to(epmc).pdf DeepDyve Pubget Overpricing