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10.1007/s40620-023-01583-2 http://scihub22266oqcxt.onion/10.1007/s40620-023-01583-2 36913081!10638190!36913081     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=36913081&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Nephrol 2023 ; 36 (8): 2375-2380 Nephropedia Template TP {{tp|p=36913081|t=2023. IgA nephropathy to proliferative glomerulonephritis with monoclonal IgAkappa deposits: a pattern switch |pdf=|usr=}}{{36913081}} gab.com Text IgA nephropathy to proliferative glomerulonephritis with monoclonal IgAkappa deposits: a pattern switch JO: J Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=36913081 #FOAvip We report the case of a 31-year-old male who presented with repeated episodes of nephritic-nephrotic syndrome in concomitance with infection. IgA was diagnosed and was initially responsive to treatment with immunosuppressors but further disease flare did not respond to treatment. Based on three consecutive renal biopsies over 8 years, a pattern switch from endocapillary proliferative IgA nephropathy to membranous proliferative glomerulonephritis with monoclonal IgAkappa deposits was observed. Bortezomib-dexamethasone combination therapy finally led to a favorable renal response. This case provides new insights into the pathophysiological mechanisms of proliferative glomerulonephritis with monoclonal immunoglobin deposits (PGNMID), highlighting the importance of repeat renal biopsies and routine evaluation of monoclonal immunoglobin deposits in proliferative glomerulonephritis with refractory nephrotic syndrome. Twit Text FOAVip IgA nephropathy to proliferative glomerulonephritis with monoclonal IgAkappa deposits: a pattern switch ????J Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=36913081 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=36913081 #FOAvip English Wikipedia <ref>{{Cite pmid|36913081}}</ref> IgA nephropathy to proliferative glomerulonephritis with monoclonal IgAkappa deposits: a pattern switch #MMPMID36913081 Li JP; Du YT; Guo C; Rao XR; Li S J Nephrol 2023[Nov]; 36 (8): 2375-2380 PMID36913081show ga We report the case of a 31-year-old male who presented with repeated episodes of nephritic-nephrotic syndrome in concomitance with infection. IgA was diagnosed and was initially responsive to treatment with immunosuppressors but further disease flare did not respond to treatment. Based on three consecutive renal biopsies over 8 years, a pattern switch from endocapillary proliferative IgA nephropathy to membranous proliferative glomerulonephritis with monoclonal IgAkappa deposits was observed. Bortezomib-dexamethasone combination therapy finally led to a favorable renal response. This case provides new insights into the pathophysiological mechanisms of proliferative glomerulonephritis with monoclonal immunoglobin deposits (PGNMID), highlighting the importance of repeat renal biopsies and routine evaluation of monoclonal immunoglobin deposits in proliferative glomerulonephritis with refractory nephrotic syndrome. |*Glomerulonephritis, IGA/complications/diagnosis/drug therapy[MESH] |*Glomerulonephritis, Membranoproliferative/diagnosis/drug therapy[MESH] |*Glomerulonephritis/pathology[MESH] |*Nephrotic Syndrome[MESH] |Adult[MESH] |Humans[MESH] |Immunoglobulin G[MESH]IgA nephropathy to proliferative glomerulonephritis with monoclonal Ig(epmc).pdf DeepDyve Pubget Overpricing