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suck abstract from ncbi


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pmid362722      West+J+Med 1978 ; 129 (4): 278-320
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  • The pathophysiology and clinical aspects of hypercalcemic disorders #MMPMID362722
  • Lee DB; Zawada ET; Kleeman CR
  • West J Med 1978[Oct]; 129 (4): 278-320 PMID362722show ga
  • FOR THE PURPOSES OF THIS REVIEW, THE VAST AND INCREASINGLY COMPLEX SUBJECT OF HYPERCALCEMIC DISORDERS CAN BE BROKEN DOWN INTO THE FOLLOWING CATEGORIES: (1) Physiochemical state of calcium in circulation. (2) Pathophysiological basis of hypercalcemia. (3) Causes of hypercalcemia encountered in clinical practice: causes indicated by experience at the University of California, Los Angeles; neoplasia; hyperparathyroidism; nonparathyroid endocrinopathies; pharmacological agents; possible increased sensitivity to vitamin D; miscellaneous causes. (4) Clinical manifestations and diagnostic considerations of hypercalcemic disorders. (5) The management of hypercalcemic disorders: general measures; measures for lowering serum calcium concentration; measures for correcting primary causes-the management of asymptomatic hyperparathyroidism.
  • |*Hypercalcemia/chemically induced/etiology/genetics/physiopathology/therapy[MESH]
  • |Adolescent[MESH]
  • |Adult[MESH]
  • |Aged[MESH]
  • |Calcium/blood[MESH]
  • |Child[MESH]
  • |Child, Preschool[MESH]
  • |Drug-Related Side Effects and Adverse Reactions[MESH]
  • |Female[MESH]
  • |Granulomatous Disease, Chronic/complications[MESH]
  • |Humans[MESH]
  • |Hyperparathyroidism, Secondary/complications[MESH]
  • |Hyperparathyroidism/complications[MESH]
  • |Hypothyroidism/complications[MESH]
  • |Infant[MESH]
  • |Infant, Newborn[MESH]
  • |Male[MESH]
  • |Middle Aged[MESH]
  • |Neoplasms/complications[MESH]


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