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10.3233/CH-221514

http://scihub22266oqcxt.onion/10.3233/CH-221514
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35754264!ä!35754264

suck abstract from ncbi


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pmid35754264      Clin+Hemorheol+Microcirc 2022 ; 82 (2): 193-198
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  • ADAMTS13 factor deficiency in severe COVID-19 may not be immune mediated - report from a pilot study #MMPMID35754264
  • George A; Deepika C; Mohan G; Srishail R; Rajendran V; Shastry S; Balakrishnan JM; Rao S
  • Clin Hemorheol Microcirc 2022[]; 82 (2): 193-198 PMID35754264show ga
  • BACKGROUND: The assessment of ADAMTS13 factor activity and inhibitor levels was conducted in severe COVID-19 patients as an observational study. RESULTS: A total of 14 patients were included and the average ADAMTS13 activity level at the time of admission was 28.54+/-30.74% (range 1.83-86.67%) which was reduced compared to controls (88.09+/-14.77). Nine patients had reduced ADAMTS13 factor activity (<40%) and 77.7% among them had severe deficiency (<10% activity). ADAMTS13 inhibitor was positive (>15 IU/mL) only in two patients and an overall mean value was 8.15+/-5.8. Elevated D-Dimer and length of hospital stay had significant correlation with ADAMTS13 activity (-0.247 and 0.306 respectively). No features of thrombotic microangiopathy were observed and hence no plasma exchange was performed. CONCLUSION: Reduced ADAMTS13 factor activity without inhibitor development may give a clue to the disease progress in COVID-19.
  • |*COVID-19[MESH]
  • |ADAMTS13 Protein[MESH]
  • |Humans[MESH]
  • |Pilot Projects[MESH]


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